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Journal of Behavioral Addictions
Authors: Nicki A. Dowling, Carrie Ewin, George J. Youssef, Stephanie S. Merkouris, Aino Suomi, Shane A. Thomas, and Alun C. Jackson

Background and aims

Few studies have investigated the association between problem gambling (PG) and violence extending into the family beyond intimate partners. This study aimed to explore the association between PG and family violence (FV) in a population-representative sample. It was hypothesized that: (a) PG would be positively associated with FV, even after adjusting for sociodemographic variables and comorbidities and (b) these relationships would be significantly exacerbated by substance use and psychological distress. A secondary aim was to explore whether gender moderated these relationships.

Methods

Computer-assisted telephone interviews were conducted with a population-representative sample of 4,153 Australian adults.

Results

Moderate-risk (MR)/problem gamblers had a 2.73-fold increase in the odds of experiencing FV victimization (21.3%; 95% CI: 13.1–29.4) relative to non-problem gamblers (9.4%; 95% CI: 8.5–10.4). They also had a 2.56-fold increase in the odds of experiencing FV perpetration (19.7%; 95% CI: 11.8–27.7) relative to non-problem gamblers (9.0%; 95% CI: 8.0–10.0). Low-risk gamblers also had over a twofold increase in the odds of experiencing FV victimization (20.0%; 95% CI: 14.0–26.0) and perpetration (19.3%; 95% CI: 13.5–25.1). These relationships remained robust for low-risk gamblers, but were attenuated for MR/problem gamblers, after adjustment for substance use and psychological distress. MR/problem gamblers had a greater probability of FV victimization, if they reported hazardous alcohol use; and low-risk gamblers had a greater probability of FV perpetration if they were female.

Discussion and conclusion

These findings provide further support for routine screening, highlight the need for prevention and intervention programs, and suggest that reducing alcohol use may be important in these efforts.

Open access

Abstract

The Madaras brickyard section found at the northernmost fringe of the Backa loess plateau is one of the thickest and best-developed last glacial loess sequences of Central Europe. In the present work high-resolution magnetic susceptibility measurements (at 2 cm) were implemented on samples from the 10 m-section corresponding to a period between 29 and 11 KY cal b2K. One aim was to compare the findings with the ice core records of northern Greenland in order to establish a high-resolution paleoclimatic record for the last climatic cycle and with findings documented in other biotic and abiotic proxies so far. Our results revealed a strong variability of loess/paleosol formation during MIS 2. Millennial time-scale climatic events that characterize the North Atlantic during the last climatic cycle have been identified. From 29 ka up to the start of the LGM, the recorded MS values show a weak, negative correlation with the temperature proxy, and a weak positive correlation with the dust concentration of Greenland. A strong correlation was observed with the local paleotemperatures. Local climatic factors must have had a more prominent effect here on loess/paleosol development than the climate shifts over Greenland. During the LGM the same pattern is seen with a stronger correlation with the dust concentrations and a weaker correlation with the local temperature. Local climatic factors, plus dust accumulation, must have had a prominent influence on loess/paleosol development here. From the terminal part of the LGM a strong positive correlation of the MS values with the temperature proxy for Greenland accompanied by a strong negative correlation with the dust concentration values is observed. Correlation with local paleotemperatures is positive and moderate, strong. Here climate shifts over Greenland, as well as local endowments equally had an important role on the development of the MS signal.

Restricted access

Including gaming disorder in the ICD-11: The need to do so from a clinical and public health perspective

Commentary on: A weak scientific basis for gaming disorder: Let us err on the side of caution (van Rooij et al., 2018)

Journal of Behavioral Addictions
Authors: Hans-Jürgen Rumpf, Sophia Achab, Joël Billieux, Henrietta Bowden-Jones, Natacha Carragher, Zsolt Demetrovics, Susumu Higuchi, Daniel L. King, Karl Mann, Marc Potenza, John B. Saunders, Max Abbott, Atul Ambekar, Osman Tolga Aricak, Sawitri Assanangkornchai, Norharlina Bahar, Guilherme Borges, Matthias Brand, Elda Mei-Lo Chan, Thomas Chung, Jeff Derevensky, Ahmad El Kashef, Michael Farrell, Naomi A. Fineberg, Claudia Gandin, Douglas A. Gentile, Mark D. Griffiths, Anna E. Goudriaan, Marie Grall-Bronnec, Wei Hao, David C. Hodgins, Patrick Ip, Orsolya Király, Hae Kook Lee, Daria Kuss, Jeroen S. Lemmens, Jiang Long, Olatz Lopez-Fernandez, Satoko Mihara, Nancy M. Petry, Halley M. Pontes, Afarin Rahimi-Movaghar, Florian Rehbein, Jürgen Rehm, Emanuele Scafato, Manoi Sharma, Daniel Spritzer, Dan J. Stein, Philip Tam, Aviv Weinstein, Hans-Ulrich Wittchen, Klaus Wölfling, Daniele Zullino, and Vladimir Poznyak

The proposed introduction of gaming disorder (GD) in the 11th revision of the International Classification of Diseases (ICD-11) developed by the World Health Organization (WHO) has led to a lively debate over the past year. Besides the broad support for the decision in the academic press, a recent publication by van Rooij et al. (2018) repeated the criticism raised against the inclusion of GD in ICD-11 by Aarseth et al. (2017). We argue that this group of researchers fails to recognize the clinical and public health considerations, which support the WHO perspective. It is important to recognize a range of biases that may influence this debate; in particular, the gaming industry may wish to diminish its responsibility by claiming that GD is not a public health problem, a position which maybe supported by arguments from scholars based in media psychology, computer games research, communication science, and related disciplines. However, just as with any other disease or disorder in the ICD-11, the decision whether or not to include GD is based on clinical evidence and public health needs. Therefore, we reiterate our conclusion that including GD reflects the essence of the ICD and will facilitate treatment and prevention for those who need it.

Open access