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Maintaining appropriate eating habits is one of the key components of good health. It is especially difficult during adolescence, a critical period in life because of the increased autonomy and the intention to take risks. Investigating the theoretical background of adolescents’ eating behaviour is therefore a worthwhile line of research. We applied the widely used health belief model to explore adolescents’ likelihood of healthy eating.

Materials and methods

A sample of adolescents (Szeged, Hungary; N = 400, age = 14–19 years; mean age = 16.01 years, SD = 1.18 years; 37% males) participated in the study. Data were collected through online, self-administered/anonymous questionnaires. Based on bidirectional correlations of the variables, we used a path analysis to examine relationships between elements of a modified health belief model.


Our modified model showed the direct impacts of cues to action, benefits, barriers, and self-efficacy, and the indirect impacts of perceived severity and susceptibility-via-cues-to-action on the likelihood of healthy eating.

Discussion and conclusions

Elements of the health belief model play a decisive role in estimating adolescents’ healthy eating behaviour. We suggest that the model can serve as a useful theoretical background in planning and evaluating prevention programs to reduce obesity and promote healthy eating.

Open access
Acta Physiologica Hungarica
H. Pikó
V. Vancsó
B. Nagy
J. Balog
M. Nagymihály
A. Herczegfalvi
L. Tímár
Z. Bán
, and
V. Karcagi

Muscular dystrophies are a genetically heterogeneous group of degenerative muscle disorders. This article focuses on two severe forms of muscular dystrophies and provides genetic data for a large cohort of Hungarian patients diagnosed within the last few years by the authors.The Duchenne/Becker muscular dystrophy (DMD/BMD) is caused by mutations in the dystrophin gene, which is located on chromosome Xp21. The genetic analysis of dystrophin is usually performed by multiplex polymerase chain reaction (PCR), which detects approximately 95% of all deletions but does not distinguish between one and two copies of the exons investigated. The present work, therefore, concentrates on the improvement of the diagnostic panel for the analysis of DMD/BMD in Hungary. Radioactively labelled cDNA probes, encompassing the whole dystrophin gene detect all the deletions and the analysis is quantitative. In addition, the new multiple ligationdependent probe amplification (MLPA) technique was recently introduced that enabled more reliable and faster quantitative detection of the entire dystrophin gene. The genomic basis of facioscapulohumeral muscular dystrophy (FSHD) is associated with contraction of the D4Z4 repeat region in the subtelomere of chromosome 4q. In case of FSHD, molecular genetic criteria still have to be improved because of the complexity of the disorder.

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