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diseased vessel endothelium. Hence, it should also be understood that such techniques are technically challenging and sometimes even unsuccessful. Overall, the incidents of dislodgement of cath-lab equipment during the procedure may occur due to
A szegedi Sebészeti Műtéttani Intézet 25 éve •
Oktatás és tudomány, eredmények 1998–2023 között
25 years of the Institute of Surgical Research, Szeged •
Education and science, achievements in 1998–2023
szakterület The role of vasodilator mediators in different types of circulatory shock (1998) kísérletes sebész-kutató Endothelial and epithelial dysfunctions in various circulatory disorders (1998) kísérletes sebész-kutató The role of endothelium
nuclei and vacuolisation in hepatocytes, ruptured endothelium of veins, sinusoidal dilatation, and necrosis criteria. The structure of the liver was normal in the rats selected for the C group. Sinusoidal dilatation and pycnotic nuclei and vacuolisation
causing increased adhesion and migration of the monocytes into the subendothelial layer causing endothelial dysfunction Anti-coagulant and anti-adhesion properties can be observed in a normal arterial endothelium however, proteolytic degradation of
2010 376 1498 1509 Narshi, C. B., Giles, I. P., Rahman, A.: The endothelium: an interface between
administration of PYC (180 mg/day) resulted in an increase in endothelium-dependent vasodilatation measured via forearm blood flow response in healthy young men ( 33 ). Improved flow-mediated dilatation following PYC administration (200 mg, 8 weeks) was also
reduced production or inactivation of endothelium-derived NO, induction of redox-sensitive transcription factors, and oxidation of critical proteins involved in vascular contractile mechanisms. Reduced NO diminishes vascular protective effects, inhibits
calcification (VC) plays an essential role in the development of cardiovascular diseases in CRF patients. VC is an active, cell-mediated process that includes vascular smooth muscle cells (VSMC) and endothelial cells [ 5, 6 ]. The interaction between endothelium
follicles, (h) the lymphocytes infiltrating the blood vessel wall, vascular endothelium and degenerated neurons of the cerebral cortex, and (i) degenerated renal tubules of the kidney Discussion Newcastle disease is an economically important disease of
human cutaneous blood perfusion signal modified by endothelium-dependent and endothelium-independent vasodilators . Microvasc. Res. 57 , 298 – 309 ( 1999 ) 10.1006/mvre.1998.2139 11
