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Melki, V., Ayon, F., Fernandez, M. és mtsa: Value and limitations of the continuous glucose monitoring system in the management of type 1 diabetes. Diabetes Metab., 2006, 32 , 123–129. Fernandez M

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Thrailkill, K. M., Liu, L., Wahl, E. C. és mtsai: Bone formation is impaired in a model of type 1 diabetes. Diabetes, 2005, 54 , 2875–2881. Wahl E. C. Bone formation is impaired in a

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Gianani, R., Eisenbarth, G. S.: The stages of type 1 diabetes. Immunol. Rev., 2005, 204 , 232–249. Eisenbarth G. S. The

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type 1 diabetes in Europe during 1989–2003 and predicted new cases 2005–20: a multicentre prospective registration study Lancet 2009 373

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Orvosi Hetilap
Authors:
Barbara Szémán
,
Géza Nagy
,
Tímea Varga
,
Anna Veres-Székely
,
Mária Sasvári
,
Dávid Fitala
,
Adrienn Szöllősi
,
Rózsa Katonai
,
Eszter Kotyuk
, and
Anikó Somogyi

reactivity in the hippocampus of murine models of type 1 diabetes: the nonobese diabetic (NOD) and streptozotocin-treated mice. Brain Res., 2002, 957 , 345–353. Gonzalez Deniselle M. C

Open access

A probiotikumok szerepe az orális egészségben.

A szájflóra Lactobacillus-összetételének vizsgálata egészséges és cukorbetegséggel élő gyermekekben

The role of probiotics in oral health.

Examination of the compound of the Lactobacillus in the oral flora in healthy and diabetic children
Orvosi Hetilap
Authors:
Anna Moldován
,
Noémi Rózsa
, and
Anna Herczegh

, Hosszúfalusi N. Environmental factors and epidemiology of childhood type 1 diabetes. [Környezeti hatások a gyermekkori kezdetű, 1-es típusú diabetes epidemiológiai változásainak hátterében.] Orv Hetil

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diabetes Type 1 diabetes (T1D) was induced with STZ (50 mg/kg; Sigma, St. Louis, MO, USA). STZ was dissolved in cold normal saline and injected immediately. According to this method, 72 h after injection, the fasting blood glucose levels were

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Mitochondrialis diszfunkció okozta betegségek

Diseases caused by mitochondrial dysfunction

Orvosi Hetilap
Authors:
Tamás Halmos
and
Ilona Suba

Monaco CM, Gingrich, MA, Hawke, T. Considering type 1 diabetes, as a form of accelerated muscle aging. Exerc Sport Sci Rev. 2019; 47: 98–107. 34 Jin S

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Abstract

Recently, an increasing amount of data on the connection between neuro-endocrine and immune systems has been gathered. Results of molecular genetic research have provided evidence for a common language of these systems including neurotransmitters, hormones and cytokines. It has been proved that the immune system is capable of producing neurotransmitters and hormones and even the endocrine system can prepare cytokines. This integrative (holistic) approach makes possible the investigation of physiological and pathological events as interactions of psychoneuro-endocrine-immune systems. The associations of autoimmune diseases and the autoimmune polyendocrine syndromes constitute a heterogeneous group of disorders characterised by decreased or lost immune tolerance against self-antigens. Molecular genetic research has explored the mechanism of the associations of diseases that are called organ-specific. Autoimmune polyendocrine syndrome type 1 is characterised by the presence of at least two of the three cardinal diseases: Addison's disease, autoimmune hypoparathyroidism and mucocutaneous candidiasis. This rare autosomal recessive syndrome is induced by mutations of the autoimmune regulator (AIRE) gene. Autoimmune polyendocrine syndrome type 2 that occurs at a much higher frequency is observed and defined as the coexistence of Addison's disease, autoimmune thyroid disease and/or type 1 diabetes mellitus. Autoimmune polyendocrine syndrome type 3 is characterised by an association of autoimmune thyroid disease and type 1 diabetes mellitus. In contrast to autoimmune polyendocrine syndrome type 1, HLA and other antigens have proved to be important in types 2 and 3 of the syndrome. Identification of genetic factors predisposing to these syndromes contributes to our understanding of the common mechanisms involved in autoimmunity and offers a possibility for early treatment and prevention as well.

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Primary immunodeficiency diseases (PIDs) are a heterogeneous group of disorders that genetically affect distinct components of the immune system; thus, predispose individuals to recurrent infections, allergy, autoimmunity, and malignancies. In this retrospective study, autoimmune diseases (ADs), which developed during the course of PID in children, were discussed.Twenty-five patients were included in this study. Symptoms related to ADs, such as autoimmune thyroiditis, type 1 diabetes mellitus, coeliac disease, juvenile idiopathic arthritis, dermatomyositis, autoimmune haemolytic anaemia, leukocytoclastic vasculitis, Henoch-Schonlein purpura, hypoparathyroidism, alopecia areata, Addison’s disease, vitiligo and systemic lupus erythematosus were detected in these patients, who have been followed with diagnosis of PID including common variable immunodeficiency, selective and partial IgA deficiency, Wiskott-Aldrich syndrome, ataxia telangiectasia, hyperimmunoglobulin E syndrome, chronic mucocutaneous candidiasis, Griscelli syndrome, and partial C4 deficiency.Immunodeficiency and autoimmune phenomenon may concomitantly present in an individual, although they seem to be incompatible ends in the spectrum of the clinical immune response. Patients with primary immune deficiency should be closely monitored for development of autoimmune diseases.

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