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activating the immune system of the hosts [ 25 ]. Just a single recent review lists many major functions for AMPs, such as direct anti-microbial effects, clearance of endotoxins, chemotaxis, modification of anti- and pro-inflammatory immune responses, wound

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, et al. Modulation of miR-155 and miR-125b levels following lipopolysaccharide/TNF-alpha stimulation and their possible roles in regulating the response to endotoxin shock. J Immunol. 2007

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., Heineman, M. J., et al.: The immune response during the luteal phase of the ovarian cycle: increasing sensitivity of human monocytes to endotoxin. Fertil. Steril., 2001, 76 (3), 555

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): Tracking the source of Bacillus thuringiensis Cry1Ab endotoxin in the environment. Biochem. Syst. Ecol. 33, 219–232. Blaise C. Tracking the source of Bacillus thuringiensis Cry1Ab

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) Copps K Human IL6 enhances leptin action in mice Diabetologia 2010 53 525 535 Spangelo BL, Judd AM, MacLeod RM, Goodman DW, Isakson PC: Endotoxin-induced release of interleukin-6

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Orvosi Hetilap
Authors: József Ongrádi, Valéria Kövesdi, and Enikő Kováts

Younes, S. A., Csire, M., Palyi, B. és mtsai: Endotoxins do not influence transplacental transmission of lymphotropic human herpesviruses and human papillomaviruses into amniotic fluid taken from healthy mothers before parturition. Acta Microbiol. et

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cytokine production by a post-transcriptional mechanism and protects against endotoxin-induced shock. J. Immunology 164, 1013-1019 (2000) Inosine inhibits inflammatory cytokine production by a post-transcriptional mechanism and

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Guo H, Wei J, Kuo PC: Nitric oxide inhibits expression of cytochrome b in endotoxin-stimulated murine macrophages. Biochem. Biophys. Res. Commun. 289, 993–997 (2001) Kuo P.C. Nitric

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, which leads to immune activation [ 52 ]. Another example is a cohort observation in PD patients, where the αSyn, and serum LPS-binding protein, endotoxin marker were increased and associated with intestinal hyperpermeability [ 53 ]. Among these studies

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Orvosi Hetilap
Authors: Olivér Rosero, Tibor Kovács, Péter Ónody, László Harsányi, and Attila Szijártó

clinical study of E5, a monoclonal antibody to endotoxin: results of a prospective, multicenter, randomized, controlled trial. The E5 Sepsis Study Group. Crit. Care Med., 1995, 23 (6), 994–1006. Warren, H. S., Amato, S

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