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]. 10 Gebistorf F, Karam O, Wetterslev J, et al. Inhaled nitric oxide for acute respiratory distress syndrome (ARDS) in children and adults. Cochrane Database Syst Rev. 2016; 2016(6): Cd002787. 10
32 346 353 Titherage MA (1997): The enzymatic measurement of nitrate and nitrite. In: Methods in Molecular Biology 100 Nitric oxide Protocols
) oxidase, xanthine oxidase/reductase, nitric oxide synthases (NOSs), etc., as well as by the depletion of cellular enzymatic and non-enzymatic antioxidants ( 5 , 12 , 30 ). In high concentrations, RONS determines the expression of hypoxia-inducible factor
) inhibition by cyclopiazonic acid (CPA) or thapsigargin ( 28 , 30 , 40 , 53 ). Both ACh and CPA increase [Ca 2+ ] i through SOCE in endothelial cells yielding nitric oxide (NO) production that eventually leads to vascular smooth muscle relaxation ( 34
Miranda, K. M., Espey, M. G. and Wink, D. A. (2001): A rapid, simple spectrophotometric method for detection of nitrate and nitrite. Nitric Oxide 5 , 62–71. Wink D. A. A rapid, simple
181 327 337 Panjwani NN, Popova L, Srivastava PK: Heat shock proteins gp96 and hsp70 activate the release of nitric oxide by APCs. J. Immunol. 168
Cancer Research (CECR), Tanta University. Biochemical analysis The level of high-density lipoproteins (HDLs) was determined according to the method described by Burstein et al. [ 13 ]. The concentration of nitric oxide
Modena, B. D., Tedrow, J. R., Milosevic, J., et al.: Gene expression in relation to exhaled nitric oxide identifies novel asthma phenotypes with unique biomolecular pathways. Am. J. Respir. Crit. Care Med
also estimated in renal tissues as per the manufacturer's protocol. The level of inducible nitric oxide synthase (iNOS) in renal tissues was determined using an ELISA kit. Histopathology of renal tissues All animals were sacrificed and the kidneys of
EC functions have been investigated. Aspirin can promote nitric oxide (NO) levels by directly stimulating the endothelial NO synthase (eNOS) [ 11 ]. Aspirin can exert protective effects on ox-LDL-induced endothelial injury by inhibiting cyclooxygenase
