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Acta Veterinaria Hungarica
Authors: Sándor Hornok, Marina Meli, Enikő Gönczi, Éva Ignits, Barbara Willi, Hans Lutz, and Regina Hofmann-Lehmann

1044 Foley, J. E. and Pedersen, N. C. (2001): ’ Candidatus Mycoplasma haemominutum’, a low-virulence epierythrocytic parasite of cats. Int. J. Syst. Evol. Micr. 51 , 815

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Acta Veterinaria Hungarica
Authors: Mikuláš Levkut, Juraj Pistl, Andrea Lauková, Viera Revajová, Robert Herich, Zuzana Ševčíková, Viola Strompfová, Renáta Szabóová, and Tatiana Kokinčáková

Salmonella serovars in faeces by specific detection of virulence genes, invA and spvC , by an enrichment broth culture-multiplex PCR combination assay. J. Clin. Microbiol. 34 , 2619–2622. Ou J. T

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Z, Erdélyi F, Fodor I: A putative latency promoter/enhancer [P(LAT2)] region of pseudorabies virus contains a virulence determinant. J. Gen. Virol. 81, 415–420 (2000) Fodor I. A

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gallolyticus subspecies: Genome plasticity, adaptation and virulence. PLoS One 6 : e20519 (2011). Hsu MT Sequencing and comparative genome analysis of two pathogenic Streptococcus

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Acta Microbiologica et Immunologica Hungarica
Authors: Aleksandra Šmitran, Dragana Vuković, Nataša Opavski, Ina Gajić, Jelena Marinković, Ljiljana Božić, Irena Živanović, Dušan Kekić, Sunčica Popović, and Lazar Ranin

antimicrobial therapy for TPh. Biofilm formation has been suggested as an important aspect of the carrier (CA) state and antibiotic treatment failures [ 3 ]. The ability of GAS to form biofilm was recently recognized as an important virulence trait of the

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Developments in Health Sciences
Authors: E Burgettiné Böszörményi, S Németh, A Fodor, K Bélafiné Bakó, D Vozik, Z Csima, and I Barcs

Amphotericin B and fluconazole. Its hypha-producing ability and enzyme production (phospholipase, protease, and haemolysin) only increase its virulence [ 6 ]. The number of C. krusei, C. glabrata , and C. tropicalis infections also increases

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Introduction Unlike Staphylococcus aureus , the negative coagulase staphylococci are not equipped with a wide range of virulence factors, and their pathogenicity is attributed to the ability of biofilm formation [ 1 , 2

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, F. C.: Virulence for mice of a proteinase-secreting strain of Candida albicans and a proteinase-deficient mutant. J Gen Microbiol 129 , 431 (1983). Virulence for mice of a proteinase-secreting strain of Candida albicans

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Introduction Multiple drug-resistant enterococci are the major cause of healthcare-associated infections due to their virulence and antibiotic resistance traits [ 1 – 4 ]. They are considered as critical agent for the

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. suis after handling animals [ 22–24 ]. Until now, more than 20 bacterial virulence-associated factors have been identified, including capsular polysaccharides (CPS), DNase (SsnA), extracellular protein factor (EPF), suilysin (Sly), serum opacity factor

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