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Mycotoxins are natural compounds that may cause various adverse toxicological manifestations in humans and animals. The nature, the severity and scope of their adverse activity are varied and in general, even in small amount they have potent carcinogenic, genotoxic effect and injure the immune system. In order to provide high level of health protection for consumers, the European Union has established strict regulatory limits, whose implementation is enforced.The EC (2001) Commission Regulation sets maximum levels for some mycotoxins in foodstuffs: for aflatoxins, ochratoxin A, patulin, deoxynivalenol, zearalenone, fumonisins, T-2 and HT-2 toxins. Particular product categories are regulated under specific decisions ordaining control of imported consignments at the point of entry. Due to the fact that only aflatoxins are addressed in the specific decisions, they are the mostly detected and notified mycotoxins in the EU Rapid Alert System for Food and Feed (RASFF). The second most frequent group, Ochratoxin A is typically detected during internal EU market controls. Most RASFF notifications concern product categories falling under specific EU decisions, especially the Aflatoxin content of nuts and nut products. Significant amount of aflatoxins can be found also in dried fruits, spices and herbs.The article reviews and analyses the data available in rapid alert system concerning mycotoxins notification, and evaluates the usefulness of this information for risk assessment. The value of RASFF system is unquestionable and it fulfils its intended function included in its name. The system is a significant source of valuable information, but for risk assessment purposes, other additional information is needed. It could be used most effectively for risk assessment, if it was to provide data on the ratio of all/tested/positive lots and if the authorities provided not only the positive results, but also the exact mycotoxins level of every analysed sample.

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6 986 994 Ji, L., Cao, K., Hu, T., Wang, S. 2007. Determination of deoxynivalenol and nivalenol chemotypes of Fusarium graminearum isolates from

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Cereal Research Communications
Authors: Szabolcs Lechoczki-Krsjak, Beáta Tóth, Csaba Kótai, Imre Martonosi, László Farády, László Kondrák, Ágnes Szabó-Hevér, and Ákos Mesterházy

. 2003. Influence of cultivar resistance, epidemic severity, and Fusarium species on the efficacy of fungicide control of Fusarium head blight in wheat and deoxynivalenol (DON) contamination of grain. Plant Disease 87 :1107

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Liu, W., Langseth, W., Skinnes, H., Elen, O., Sundheim, L. 1997. Comparison of visual head blight ratings, seed infection levels and deoxynivalenol production for assessment of resistance in cereals inoculated with Fusarium culmorum. European J. Plant

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, R., Schuhmacher, R., Grausgruber, H., Ruckenbauer, P. (2003) Fusarium head blight reactions and accumulation of deoxynivalenol moniliformin and zearalenone in wheat grains, Cereal Research Communications 31: 407

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Bai, G.H., Desjardins, A.E., Plattner, R.D. 2001. Deoxynivalenol-nonproducing Fusarium graminearum causes initial infection, but does not cause disease spread in wheat spikes. Mycopathologia 153: 91

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., Schollenberger, M. and Karlovsky, P. (2004): Microbial detoxification of mycotoxin deoxynivalenol. J. Basic Microbiol. 44, 147-156. Microbial detoxification of mycotoxin deoxynivalenol J. Basic Microbiol

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dose rate of azoxystrobin and metconazole on the development of Fusarium head blight and the accumulation of deoxynivalenol (DON) in wheat grain. Eur. J. Plant Pathol., 108: 469–478. Jenkinson P

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., Desjardins, A. E., Plattner, R. D. (2001) Deoxynivalenol-nonproducing Fusarium graminearum causes initial infection, but does not cause disease spread in wheat spikes. Mycopathologia 153 , 91-98. Deoxynivalenol

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Res. Commun., Suppl. B 36 : 31 – 35 . Cowger , C. , Arrellano , C. 2010 . Plump kernels with high deoxynivalenol linked to late Gibberella zeae infection and marginal

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