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Vallega, V. 1995. Triticum monococcum: bread baking quality, amino acid composition and toxicity in coeliac disease. Annual Wheat Newsletter 41. Vasil, I.K., Vasil, V. 1999. Transgenic cereals: Triticum

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patients with hypogammaglobulinemia. Oral Surg Oral Med Oral Pathol Oral Radiol. 2012; 114: e19–e24. 31 Muñoz F, Del Río N, Sóñora C, et al. Enamel defects associated with coeliac

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Orvosi Hetilap
Authors: Aliz Bohner-Beke, Anikó Kőnigné Péter, Lívia Vass, Szabolcs Halasi, János Kránicz, and Henriette Pusztafalvi

–88. [Hungarian] 16 Rigó A, Nagy D, Bíró M, et al. Quality of life in coeliac disease. [Életminőség lisztérzékenységben.] Alkalmazott Pszichológia 2014; 14: 117–131. [Hungarian

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Orvosi Hetilap
Authors: Ákos Iliás, Fióna Petra Rózsa, Lóránt Gönczi, Barbara Dorottya Lovász, Zsuzsanna Kürti, and Péter László Lakatos

intolerance. J Pediatr. 2016; 92: 486–492. 34 Balamtekin N, Baysoy G, Uslu N, et al. Fecal calprotectin concentration is increased in children with celiac disease: relation with

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. S., Venanzi, E. S., Klein, L., et al.: Projection of an immunological self shadow within the thymus by the aire protein. Science, 2002, 298 (5597), 1395–1401. Sollid, L. M.: Molecular basis of celiac disease

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-analysis. QJM, 2016, 109 (7), 461–466. 48 Krauss, N., Schuppan, D.: Monitoring non-responsive patients who have celiac disease. Gastrointest. Endosc. N. Am., 2006

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with their monozygotic twin [ 11 ]. Alterations in intestinal flora have been hypothesized to contribute in the development of several other autoimmune diseases, including celiac disease, allergy, multiple sclerosis, rheumatoid arthritis, and ankylosing

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Tiittanen, M., Westerholm-Ormio, M., Verkasalo, M., et al.: Infiltration of forkhead box P3-expressing cells in small intestinal mucosa in coeliac disease but not in type 1 diabetes. Clin. Exp. Immunol., 2008, 152 , 498

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coeliac disease. Lancet, 356 (9227): 399–400. Genazzani, A. D., Bersi, C., Luisi, S., Fruzzetti, F., Malavasi, B., Luisi, M., Petraglia, F., Genazzani, A. R. (2001): Increased adrenal steroid secretion in response to CRF in

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mucosal vitamin C concentrations are highly reduced [ 56 ]. Furthermore, when applying vitamin C to duodenal explants derived from patients suffering from coeliac disease due to a hypersensitivity reaction to wheat gliadin and similar proteins from rye and

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