Panton–Valentine leukocidin ( pvl ) [ 4 ]. pvl is one of the important virulence factors in S. aureus and is composed of two protein components S (38 KDa) and F (32 KDa), which are controlled by the Luks/pv and Lukf/pv genes. The product of this
39 260 Czirók, É., Marton, A., Csík, M., Solt, M.: Virulence factors of plasmid profile detertmination and genetic probing for Shiga-like toxins of Escherichia
) 32. Pricope-Ciolacu L , Nicolau AI , Wagner M , Rychli K : The effect of milk components and storage conditions on the virulence of Listeria monocytogenes as
, tet M), chloramphenicol [ cat (pC221), cat (pC194), cat (pC223)], and aminoglycosides [ aac(6′)-Ie-aph(2″)-Ia, aph(3′)-IIIa ] as well as genes encoding virulence factors: enterotoxins ( se A, se B, se C, se D, se E), shock syndrome toxin ( tst
predator–prey system could be present. Virulence is used to be mentioned in pathogenic bacteria or protozoa. These have virulence factors, which stimulate their infectivity and aggressivity. Tetrahymena itself, in general, is not pathogenic; however
Tóth, I., Schmidt, H., Kardos, G., Lancz, Z., Creuzburg, K., Damjanova, I., Pászti, J., Beutin, L. and Nagy, B. (2009): Virulence genes and molecular typing of different groups of Escherichia coli O157 strains in cattle. Appl. Environ. Microbiol. 75
significance in Parkinson’s disease . FASEB J. 23 , 3263 – 3272 . 28. Mayer , F. L. , Wilson , D. , Hube , B. ( 2013 ) Candida albicans pathogenicity mechanisms . Virulence
continued process, the chronic gastritis is created. These conditions in different patients may lead to gastric atrophy, intestinal metaplasia, or gastric cancer, depending on virulence factors, host genetic factors and, as previously stated, the host immune
) 27. Wassenaar TM , Zschüttig A, Beimfohr C , Geske T , Auerbach C , Cook H , Zimmermann K , Gunzer F : Virulence genes in a probiotic E. coli product with a
The virulence frequency of 750 wheat powdery mildew isolates of wheat genotypes, carrying 23 Pm-genes and gene combinations, was studied over ten consecutive years from 2004 to 2013. Seventy-eight previously known and 39 new pathotypes were identified during this period. The results indicate that the majority of Pm-genes have high level of virulence. Sixty to ninety percent of the isolates were virulent to Pm6, Pm8, Pm8+11, Pm2+4b+8, Pm3g, Pm10+15, Pm10+14+15. The virulence frequency was variable for Pm1a, Pm2, Pm3a, Pm3b, Pm3c, Pm5, Pm7 genes and reached high level in certain years. The virulence frequency to genes Pm20, Pm37, Pm4a+ and to gene combination with Pm3c+5a+35 and breeding lines CN240/06, CN98/06 and CN158/06 ranged from 1 to 8%. Bread wheat lines CN240/06, CN98/06 and CN158/06, derived from interspecific crosses, proved to be highly resistant to powdery mildew.
