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  • Author or Editor: A. Weidner x
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A leaf rust resistance gene transferred from the tetraploid wheat Triticum timopheevii (Zhuk.) Zhuk. (genomic composition: A t A t GG) into common wheat Triticum aestivum L. conditioned resistance at the seedling and adult plant stages in the introgression line ‘line 842-2’. To determine chromosome location and to map the resistance gene an F 2 population from a cross between ‘line 842-2’ and susceptible wheat cultivar ‘Skala’ was developed and screened against leaf rust pathotype 77 ( Puccinia triticina Erikss.). Microsatellite markers detected introgressions of the T. timopheevii genome on chromosomes 1A, 2A, 2B, 5B and 6B of ‘line 842-2’. Linkage analysis revealed an association between leaf rust resistance and microsatellite markers located on chromosome 5B. The markers Xgwm880 and Xgwm1257 were closely linked to the resistance gene with genetic distances of 7.7 cM and 10.4 cM, respectively. Infection type tests with three leaf rust isolates resulted in different patterns of infection types of ‘line 842-2’ and ‘Thatcher’ near-isogenic line with the Lr18 gene on chromosome 5B. The data corroborated the hypothesis of the diversity of the resistance coming from T. timopheevii . The resistance gene of the introgression ‘line 842-2’ seems to be different than Lr18 and therefore it was designated LrTt2 .

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Cereal Research Communications
Authors: S. Navakode, A. Weidner, R. Varshney, U. Lohwasser, U. Scholz, and A. Börner

Barley is more sensitive than the other major cereal crops to aluminium (Al) toxicity. Here, a doubled haploid mapping population was exploited to study the inheritance of Al tolerance in barley. Quantitative trait locus (QTL) mapping was based on a genetic map constructed with genic markers. Al tolerance QTL were identified on chromosomes 2H, 3H and 4H. A sequence homology search was used to derive the putative function of the genes linked to the QTL, in order to identify potential candidate genes for Al tolerance. Some of these candidates are implicated in stress/defence responses, in particular, stress signal transduction, transcription regulation factors and cell metabolism.

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