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., Murad, F., Burnstock, G. (1992) Colocalization of nitric oxide synthase and NADPH-diaphorase in the myenteric plexus of the rat gut. Neurosci. Lett. 143 , 60–64. Burnstock G

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Pataricza J, Tóth GK, Penke B, Höhn J, Papp JGy: Effect of selective inhibition of potassium channels on vasorelaxing response to cromakalim, nitroglycerin and nitric oxide of canine coronary arteries. J. Pharm

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localization of neuronal nitric oxide synthase in the rat nucleus of the solitary tract in relation to vagal afferent inputs . Neuroscience 118 , 115 – 122 . 6. Baisden , R. H

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References 1. Bicker , G. , Schmachtenberg , O. , De Vente , J. ( 1996 ) The nitric oxide/cyclic GMP messenger system in olfactory pathways of the locust

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Journal of Thermal Analysis and Calorimetry
Authors: L. Chmielarz, M. Zbroja, P. Kuśtrowski, B. Dudek, A. Rafalska-Łasocha and R. Dziembaj

Abstract  

Alumina, zirconia and titania pillared montmorillonites additionally modified with silver were tested as catalysts of NO reduction with NH3 or C2H4. Ammonia was much more effective reducer of NO than ethylene. The silver containing TiO2-pillared clay has been found to be the most active catalyst for NO reduction both with NH3 or C2H4. Oxidation of the reducing agents by oxygen limited the NO conversion in the high temperature region. The ammonia and nitric oxide adsorption sites were studied by the temperature programmed desorption methods (TPD).

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Acta Physiologica Hungarica
Authors: Z Oreščanin, Z Oreščanin, Z Oreščanin, SR Milovanović, SR Milovanović and SR Milovanović

In the present study we investigated the mechanism of nitric oxide induced relaxation of renal arteries, with or without endothelium, taken from normotensive and spontaneously hypertensive (SH) rats. With this purpose in mind, the effects of the nitric oxide donor, sodium nitroprusside (SNP), with and without L-arg in the medium, on isolated rat renal artery relaxation were studied. Relaxing effect of SNP was higher in normotensive (10-5 M of SNP caused 220% of relaxation in the cases with endothelium and 240% without endothelium), in comparison with SH rats (100% of relaxation with endothelium and 150% without). L-arg antagonized the relaxing effect of SNP in the examined renal arteries, more in normotensive (100-160% with endothelium and 110-195% without) than in hypertensive ones (0-10% with endothelium and 35-75% without) at SNP concentrations 10-7-10-5 M, respectively (*P<0.05; **P<0.001). L-arg did not significantly change relaxing effect of SNP in the isolated renal arteries with endothelium taken from SH rats, which show that L-arg, by modifying the chemical versatility of NO into redox active forms -nitrosonium (NO+) and -nitroxyl (NO-), produces different relaxing effects in normotensive and hypertensive isolated arteries of rats, with or without endothelium, potentiating the role of nitroxyl induced relaxation in SH rats.

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Abstract  

The long-term goal of this investigation is to study the effects of increased cholesterol levels on the molecular activity of membrane-bound enzymes such as nitric oxide synthase, that are critical in the functioning of the cardiovascular system. In this particular investigation, we used differential scanning calorimetry (DSC) and dielectric thermal analysis (DETA) to study the effect of added cholesterol on melting/recrystallization and dielectric behavior, respectively, of phosphatidylcholine (PC) bilayered thin films. We also used electrochemical methods to investigate the effect of added cholesterol on the redox behavior of the oxygenase domain of nitric oxide synthase as a probe embedded in the PC films. The results show that added cholesterol in the PC films seems to depress the molecular dynamics as indicated by lowered current responses in the presence of cholesterol as well as a slight increase of the transition temperature in the overall two-phase regime behavior observed in PC–cholesterol films. These results are rationalized in the context of the general DSC and DETA behaviors of the PC–chol films.

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References Bing , R. J. , Miyataka , M. , Rich , K. A. , Hanson , N. , Wang , X. , Slosser , H. D. and Shi , S. R. ( 2001 ): Nitric oxide, prostanoids

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–332. 2 Moncada S. Nitric oxide. J Hypertens Suppl. 1994; 12: S35–S39. 3 Brown MA, Magee LA, Kenny LC, et al. The hypertensive disorders of

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Bagi Z, Koller A: Lack of nitric oxide mediation of flow-dependent arteriolar dilation in type I diabetes is restored by sepiapterin. J. Vasc. Res. 40, 47–57 (2003

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