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Acta Physiologica Hungarica
Authors: Z Kojić, Z Kojić, Z Kojić, Lj Šćepanović, Lj Šćepanović, Lj Šćepanović, N Popović, N Popović and N Popović

In our previous work we have shown that in mouse heart basal level of endothelial produced nitrite, as a marker of nitric oxide (NO) formation, was 9.7 nmol l-1. Bradykinin (10 mmol l-1) induced a 5-fold rise in nitrite release, the coronary venous effluent concentration being 58 nmol l-1, but there was no effect on myocardial oxygen consumption (MVO2). The aim of this study was to assess the levels of authentic nitric oxide solution, exogenously applied, on myocardial oxygen consumption. Isolated mouse hearts (n=36) were paced (500 imp./min) and perfused at constant flow (16.0±0.3 ml g-1 min-1). When coronary vasculature resistance was carefully controlled by adenosine (1 mmol l-1), authentic nitric oxide solution, in a concentration less than 5 mmol l-1 did not alter myocardial oxygen consumption. Only concentrations of nitric oxide higher than 5 mmol l-1 induced reduction in myocardial oxygen consumption. Thus in the saline perfused mouse heart, with carefully controlled vasodilatation, modulating myocardial nitric oxide levels using an arterial application of authentic nitric oxide, concentrations higher than 5 mmol l-1 of nitric oxide were required to induce a decrease in myocardial oxygen consumption.

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Procedures for the Online and Offline Measurement of Exhaled Lower Respiratory Nitric Oxide and Nasal Nitric Oxide, 2005. Am. J. Respir. Crit. Care Med., 2005, 171, 912–930. Silkoff, P. E., Carlson, M

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Molnár, M., Sütö, T., Tóth, T. és mtsa: Prolonged blockade of nitric oxide synthesis in gravid rats produces sustained hypertension, proteinuria, thrombocytopenia, and intrauterine growth retardation. Am. J. Obstet. Gynecol., 1994, 170 , 1458

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Acta Biologica Hungarica
Authors: B. Mornagui, A. Grissa, M. Duvareille, C. Gharib, A. Kamoun, S. El-Fazaa and N. Gharbi

Akesson, B., Henningsson, R., Salehi, A., Lundquist, I. (1999) Islet constitutive nitric oxide synthase and glucose regulation of insulin release in mice. J. Endocrinol. 163, 39

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., Desikan, R., Hurst, R. D., Hancock, J. T., Neill, S. J. (2000) NO way back: nitric oxide and programmed cell death in Arabidopsis thaliana suspension cultures. Plant J. 24 , 667-677. NO way back: nitric oxide and programmed

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31 840 Koh, H. Y., Jacklet, J. (1999) Nitric oxide stimulates cGMP production and mimics synaptic responses in metacerebral neurons of Aplysia. J. Neurosci. 19 , 3818

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Introduction In the past decade, the nitric oxide (NO) effect on plants metabolism has been given much consideration [ 1 , 2 ]. NO is a short-lived free radical that diffuses through membranes. NO is found in different plant

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Blottner, D., Grozdanovic, Z., Gossrau, R. (1995) Histochemistry of nitric oxide synthase in the nervous system. Histochem. J. 27 , 785–811. Gossrau R

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} Endocrinology 2004 145 5021 Baratti CM, Kopf SR: A nitric oxide synthase inhibitor impairs memory

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196 204 Antus B, Barta I, Horvath I, Csiszer E: Relationship between exhaled nitric oxide and treatment response in COPD patients with exacerbations. Respirology 15, 472–477 (2010) Csiszer E

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