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/g in the liver). As a consequence of methylone use, striated heart muscle damage was observed. Steatosis of the heart muscle, congenital heart disease, and bronchial asthma were listed as potential facilitators of the drug-induced death, with relatively

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than in control subjects. Interestingly, there was a difference in FGF-2 concentrations according to etiologies of PH, with the highest levels seen in patients with primary PAH compared to patients with congenital heart disease or connective tissue

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