Authors:Farideh Rahmani, Abbas Fooladi, Seyed Marashi and Mohammad Nourani
Akond, M. A., Alam, S., Hasan, S. M. R., Uddin, S. N., Shirin, M.: Antibiotic resistance of Vibrio cholerae from poultry sources of Dhaka, Bangladesh. Advances in Biological Research 2 , 60–67 (2008).
Authors:Hagen Frickmann, Andreas Hahn, Stefan Berlec, Johannes Ulrich, Moritz Jansson, Norbert Georg Schwarz, Philipp Warnke and Andreas Podbielski
], resistance against bactericidal first-line drugs like beta-lactam antibiotics, caused by enzymes like, e.g., extended spectrum beta-lactamases (ESBL) or carbapenems for E. coli , as well as by penicillin-binding proteins for methicillin-resistant S. aureus
Field experiments were carried out with soybean [
(L.) Merrill] on a Haplic Chernozem soil. Eleven treatment combinations were applied with increasing rates of fertilizers in three replicates. At full maturity the dry weight production, total biomass production, root weight, yield, shoot NPK concentration and severity of
infection were determined. The highest degree of
infection was found in the lowest NPK treatment, while the lowest rate of disease was observed for the highest NPK combination. By increasing the NK supply, the degree of infection decreased. At the same fertilizer rates, significantly lower infection was observed at higher phosphorus levels. The dry weight production, total biomass production of the shoots, pod weight and nitrogen concentration of the shoots were negatively correlated with the rate and severity of infection, which was positively correlated with the concentration of phosphorus. No correlation was found between root weight and infection or between potassium concentration and infection.
Buerstmayr, H., Lemmens, M., Berlakovich, S., Ruckenbauer, P. (1999) Combining ability of resistance to head blight caused by
(W. G. Smith) in the F1 of a seven parent diallel of winter wheat (
L.), Euphytica 110
Authors:Fabrizio Bertelloni, Claudia Salvadori, Giulia Lotti, Domenico Cerri and Valentina Virginia Ebani
, domestic dogs, even though without clinical forms, may excrete enterococci in their feces contaminating the environment shared by humans.
Enterococci are of particular concern for their intrinsic antibiotic resistance, particularly to cephalosporins