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, Bramanti TE. Factors in virulence expression and their role in periodontal disease pathogenesis. Crit Rev Oral Biol Med. 1991; 2: 177–281. 36 Nakou M, Kamma JJ, Andronikaki A, et

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Med. 2007; 35: 1284–1289. 11 Bassetti M, Righi E, Carnelutti A. Bloodstream infections in the Intensive Care Unit. Virulence 2016; 7: 267–279. 12 Averbuch D

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Acta Microbiologica et Immunologica Hungarica
Authors: Fatima Bachir Halimeh, Rayane Rafei, Seydina Diene, Mary Mikhael, Hassan Mallat, Marcel Achkar, Fouad Dabboussi, Monzer Hamze and Jean-Marc Rolain

(enteroinvasive E. coli ) share similar genetic (analogous virulence plasmid) and biochemical features [ 7 ]. Unlike E. coli , Shigella isolates are less active biochemically and react with limited set of antisera and shared with EIEC pathogenicity genes [ 8

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Acta Veterinaria Hungarica
Authors: Rossana Abreu, Cristobalina Rodríguez-Álvarez, María Lecuona, Beatriz Castro-Hernández, Juan Carlos González, Armando Aguirre-Jaime and Ángeles Arias

. , Haesebrouck , F. and De Vliegher , S. ( 2014 ): Effect, persistence, and virulence of coagulase-negative Staphylococcus species associated with ruminant udder health . J. Dairy Sci. 97 , 5275 – 5293 . doi: 10.3168/jds.2013

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. Chiers , K. , de Waele , T. , Pasmans , F. , Ducatelle , R. and Haesebrouck , F. ( 2010 ): Virulence factors of Actinobacillus pleuropneumoniae involved in colonisation, persistence and induction of lesions in its porcine host . Vet. Res

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and airway cytokines while increasing the virulence of colonizing bacteria. J Mol Med. 2016; 94: 667–679. 24 Pikaljuk VS, Salanin VV, Zsuravel EA, et al. Structural changes in

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Orvosi Hetilap
Authors: Bálint Gergely Szabó, Katalin Szidónia Lénárt, Béla Kádár, Andrea Gombos, Balázs Dezsényi, Judit Szanka, Ilona Bobek and Gyula Prinz

eredményei, 2013.] [Hungarian] 21 Blumental, S., Granger-Farbos, A., Moïsi, J. C., et al.: Virulence factors of Streptococcus pneumoniae. Comparison between African and

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Orvosi Hetilap
Authors: Márton Koch, Katalin Tímea Török, Ferenc Nagy, Viktor Soós, Éva Pozsgai, Zsuzsanna Lelovics, Anna Nagy and Csaba Varga

. 16 Kuczmog Z. Examination of virulence markers of West Nile virus. [A nyugat-nílusi vírus virulencia markereinek vizsgálata.] Thesis. Budapest, 2014. [Hungarian

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Orvosi Hetilap
Authors: Gergely György Nagy, Zsuzsa Tudlik, Lajos Gergely, József Kónya, Piroska Orosi, Éva Rákóczi, Judit Szabó, Csaba Várvölgyi, Eszter Vitális and György Paragh

virulence and zoonotic potential of Streptococcus suis . Sci Rep. 2016; 6: 28984. 4 Liu H, Gao XY, Fu SH, et al. Molecular evolution of emerging

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The association between putative virulence genes in Campylobacter jejuni clinical isolates, in vitro invasive capability and severity of infection is yet to be clearly described. We have characterized three virulence genes and correlated their presence with the severity of infection and in vitro invasiveness. We studied eight C. jejuni strains isolated from patients whose clinical data were scored to determine severity of infection. Cytolethal distending toxin (cdtB) , invasion associated marker (iam) and Campylobacter invasion antigen (ciaB) genes were detected by PCR and INT407 cells used for invasion assays. Two strains positive for all three genes were the most invasive and isolated from patients with the most severe infection. Four strains positive for two genes and two strains negative for all the three genes were identified. The two cdtB + ve / ciaB + ve strains were more invasive than the cdtB + ve / iam + ve strains. One of the cdtB ve / ciaB ve strains showed invasion levels similar to cdtB + ve / ciaB + ve strains, but the second strain had a non-invasive phenotype. The findings indicate a correlation between in vitro invasive capability, and the presence of all three genes. The pattern of association between invasiveness and molecular characterization suggests that the ciaB gene confers a more invasive capability.

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