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Kita, Y., Aranami, Y., Aranami, Y., Nomura, Y., Johnson, K., Wakabayashi, T., Fukunishi, I.: Japanese organ transplant law: a historical perspective. Prog. Transplant., 2000, 10 , 106
ovarian steroid secretion in Japanese quail (in Hungarian, with English abstract). Magyar Állatorvosok Lapja 50, 353-355. Richter, G. (1995): Inkorporation und Mobilisierung von Vitamin A bei Legehennen. Arch. Anim
this study was to examine the anthropometric, body composition, and somatotype characteristics of Japanese young women and to focus on normal-weight obesity syndrome and sarcopenia diagnosis criteria. Methods
794 Tanaka, F., Takeuchi, T., Tanii, A., Miyajima, K., Abe, H., Kuninaga, S.: A causal pathogen of scab on potato Streptomyces turgidiscabies n.sp (In Japanese). Ann Phytopathol Soc Jpn 61
. 136 757 761 Kerti, A. and Bárdos, L. 1999: Storage of retinoids and beta-carotene in the genital organs of Japanese quail. Acta Vet. Hung. 47 , 95
Asem, E., Marrone, B. L. and Hertelendy, F. (1985): Steroidogenesis in ovarian cells of the Japanese quail ( Coturnix coturnix japonica ). Gen. Comp. Endocrinol. 60 , 353-360. Steroidogenesis in ovarian cells of the Japanese
muscle actin (α-SMA) (Invitrogen, Life Technologies Japan Ltd., Japan; ready-to-use) and desmin (Dako; diluted 1:100). Antigen retrieval was performed via heating in 10 mm sodium citrate acid-buffered solution at 121 °C for 15 min. IHC was conducted using
sit-up performance was associated with sarcopenia status and prevalence of sarcopenia diagnostic criteria. Methods Subjects A total of 93 Japanese older women aged 53–78 years were recruited through
in broiler chickens (in Japanese, with English abstract). J. Jpn. Vet. Med. Assoc. 51 , 528-532. An outbreak of cholangiohepatitis due to Clostridium perfringens in broiler chickens (in Japanese, with English abstract
The effect of carbendazim, a widely applied cereal seed dressing agent, was studied in Japanese quail (Coturnix coturnix japonica)during an overall period of eight weeks, consisting of a four-week feeding phase and a subsequent four-week excretion period. Body mass and feed consumption of the birds were monitored and residues of the active ingredient were determined by an analytical chemical method. During the eight-week study period, changes (either decrease or increase) directly attributable to the toxic effects of carbendazim were not found either in body mass or in feed consumption. Active ingredient levels exceeding the limit of detection were found in the liver (average: 0.0262 mg/kg) and pectoral muscle (average: 0.0236 mg/kg) of the birds and also in the egg (0.0338 mg/kg) samples. From the results it can be concluded that through the consumption of cereal seeds dressed with carbendazim, this active ingredient can be incorporated into the tissues of animals. Via the food chain, carbendazim can also enter the human organism, where it may cause various pathological changes in interaction with other chemicals.