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fixed-effects analysis model was used (Q: p > 0.1 and I 2 ≤ 50% or Q: p ≤ 0.1 and I 2 < 25%). For the association between T. gondii types and outcomes of human infection, the odds of a given T. gondii type were compared in the case group
pooled OR estimate of each study was calculated by the fixed-effects model (the Mantel–Haenszel method), otherwise the random effects model (the DerSimonian–Laird method) was utilized. Furthermore, to explore the source of between-study heterogeneity, the
pooled OR estimate of each study was calculated by the fixed-effects model (the Mantel–Haenszel method), otherwise the random effects model (the DerSimonian–Laird method) was utilized. Furthermore, to explore the source of between-study heterogeneity, the
