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Bogdan, C. (2001): Nitric oxide and the regulation of gene expression. Trends Cell Biol. , 11 , 66–75. Bogdan C Nitric oxide and the regulation of gene expression

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Some studies suggested a positive effect against cardiometabolic diseases of supplementation of alpha-linolenic acid (ALA, C18:3 n-3) rich foods in pathological subjects, even if the total literature is controversial. In order to clarify ALA-rich chia seed action in hypertensive model with the overt pathology and without drug interference, in the present study the biochemical markers of cardiometabolic diseases (endothelin-1, ET-1; nitric oxide, NO; and bradykinin, BK) in Spontaneously Hypertensive Rats (SHRs) were analysed after 5% chia seed dietary supplementation for five weeks, and compared with the staple raw material wheat and corn. At the end of the experimental period, also plasma antioxidant capacity and inflammatory condition were evaluated. Our results showed that the chia seed group was more oxidized. On the other hand, ET-1 significantly decreased in chia seed group, and there was no difference between groups for NO, BK, and the inflammatory C-reactive protein (CRP). In conclusion, some positive effects of chia seed consumption on cardiometabolic markers in SHRs were observed, despite this the association of chia seeds with antioxidants is suggested to avoid plasma oxidation increase.

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Abstract

Preparation of a Fe-mordenite catalysts was carried out by impregnation using Fe(acac)3 precursor in order to have iron oxide species deposited at the surface of the zeolite. The selective presence of iron oxide species was determined and ascertained by temperature programmed reduction (TPR). In the selective catalytic reduction of NO by ammonia, no difference of conversion between the catalysts was observed indicating that well dispersed iron oxide species are active species for this reaction. Nevertheless, the obtained activity remains lower than catalysts containing iron cationic species at the exchange sites.

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between nitric dioxide ( • NO 2 ), carbonate radical anion (CO 3 •− ) and hydroxyl radical ( • OH) in the production of luminol radicals, (iv) the contribution of peroxynitrous acid (ONOOH) homolysis, and (v) that of nitric oxide ( • NO) oxidation to the

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Sohn Verlag , 2007 , p. 1032 . [13] R. Z. Kavtaradze , D. O. Onishchenko , A. A. Zelentsov , and S. S. Sergeev , “ The influence of rotational charge motion intensity on nitric oxide formation in gas-engine cylinder ”, Int. J. Heat Mass

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., Golden, M. & Benjamin, N. (1995): Chemical generation of nitric oxide in the mouth from the enterosalivary circulation of dietary nitrate. Nat. Med. , 1, 546–551. Benjamin N. Chemical

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., Tanimoto M. Removal processes of nitric oxide along positive streamers observed by laser-induced fluorescence imaging spectroscopy, Chemical Physics Letters , Vol. 323, 2000, pp. 542–548. Tanimoto M

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Acta Alimentaria
Authors: P. Sipos, K. Hagymási, A. Lugasi, Edit Fehér, F. Örsi and A. Blázovics

irritation stimulates intracolonic nitric oxide release in humans. Gut , 38 (5), 719-23. Bile acid induced colonic irritation stimulates intracolonic nitric oxide release in humans Gut

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56 723 728 Grider, J.R. (1993): Interplay of VIP and nitric oxide in regulation of the descending relaxant agents in isolated gastric

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oshimizu , K. & O higashi , H. ( 2003 ): Synergistic suppression of superoxide and nitric oxide generation from inflammatory cells by combined food factors . Mutat. Res ., 523–524 , 151 – 161 . N

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