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Hungarian Medical Journal
Authors:
Anna Körner
,
Péter Tóth-Heyn
,
Antal Dezsőfi
,
Gábor Veres
,
László Madácsy
, and
András Arató

, E. B. et al.: Thyroid autoimmunity in children and adolescents with type 1 diabetes. Diabet. Care, 2002, 25 , 1346–1350. Lang E. B. Thyroid autoimmunity in children and

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Orvosi Hetilap
Authors:
Anna Körner
,
Péter Tóth-Heyn
,
Antal Dezsőfi
,
Gábor Veres
,
László Madácsy
, and
András Arató

, E. B. és mtsai: Thyroid autoimmunity in children and adolescents with type 1 diabetes. Diab. Care, 2002, 25 , 1346–1350. Lang E. B. Thyroid autoimmunity in children and

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Soltész, G., Madácsy, L., Békefi, D. és mtsa: Rising incidence of type 1 diabetes in Hungarian children (1978–1987). Diabet. Med., 1990, 7 , 111–114. Békefi D. Rising incidence of type

Open access

Gyürüs, E., Green, A., Patterson, C. C. és mtsa: Dynamic changes in the trends in incidence of type 1 diabetes in children in Hungary (1978–98). Pediatr. Diabetes, 2002, 3 , 194

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mental health [ 5 , 6 ]. In Hungary, the sensor has only recently become widely available for adults with type 1 diabetes (T1D), since it has been subsidized by health insurance. Thus, there is not yet much feedback on satisfaction with the sensor and

Open access

Patterson, C. C., Dahlquist, G. G., Gyürüs, E, Green, A., Soltész, Gy. and the EURODIAB Study Group: Incidence trends for childhood type 1 diabetes in Europe during 1989–2003 and predicted new cases 2005-20: a multicentre peospective registration study

Open access

Incidence of type 1 diabetes (T1D) is on the rise and yet, despite decades of research, the exact ethology of the disease still remains a mystery. The autoimmune reaction, which ultimately leads to the destruction of pancreatic beta cells, causing insulin deficiency and T1D, is a result of genetic susceptibility and environmental factors. Precisely, what are these environmental factors? Current popular opinion implies these pathogens, such as viruses, especially human enteroviruses, are a triggering factor. On the other hand, the hygiene hypotheses states in which the increase of autoimmune diseases, such as T1D, can, in fact, be explained by the decrease of infections, and infectious agents, more like viruses, actually serve as a defense mechanism, therefore, protect us from developing certain autoimmune diseases. Additionally, the relationship between the gut microbiota and autoimmune diseases is currently gaining increased interest including relative research now demonstrating how the guts immune system plays a crucial role in the development of autoimmune diseases. This literature review aims to evaluate these three popular suspects: Viral infections, hygiene and gut microbiota, in relation to their potential triggering effect on T1D and their close relationship to one another.

Open access

Kis, J., Engelmann, P., Heyam, J., et al.: The possibility of immunological prevention in type 1 diabetes mellitus. [Az immunológiai prevenció lehetősége 1-es típusú diabetes mellitusban.] LAM, 2006

Open access

Varga, T., Somogyi, A., Firneisz, G. és mtsai: Higher serum dipeptidyl peptidase-4 activity in type 1 diabetes mellitus than type 2: a direct comparison. Diabetologia, 2008, 51 , S239, A589. Firneisz G

Open access
Orvosi Hetilap
Authors:
Judit Nádas
,
Zsuzsanna Putz
,
József Fövényi
,
Zsolt Gaál
,
András Gyimesi
,
Tibor Hídvégi
,
Nóra Hosszúfalusi
,
Gyula Neuwirth
,
Tamás Oroszlán
,
Pál Pánczél
,
Győző Vándorfi
,
Gábor Winkler
,
István Wittmann
, and
György Jermendy

Kilpatrick, E. S., Rigby, A. S., Atkin, S. L.: Insulin resistance, the metabolic syndrome, and complication risk in type 1 diabetes: „double diabetes” in the Diabetes Control and Complications Trial. Diabet. Care, 2007, 30 , 707

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