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Orvosi Hetilap
Authors: Árpád Czifra, Alida Páll, Veronika Sebestyén, Kitti Barta, István Lőrincz, József Balla, György Paragh, and Zoltán Szabó

disorders and heart disease. In: Braunwald, F. (ed.): Heart Disease. 5th ed. W. B. Saunders Co., Philadelphia, 1997. Meier, P., Vogt, P., Blanc, E.: Ventricular arrhythmias and sudden cardiac death in end-stage renal

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Orvosi Hetilap
Authors: Kitti Barta, Zoltán Szabó, Csaba Kun, Csaba Munkácsy, Tünde Magyar, Orsolya Bene, László Csiba, and István Lőrincz

55 255 256 Guilleminault, C., Connolly, S. J., Winkle, R. A.: Cardiac arrhythmia and conduction disturbances during sleep in 400

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1110 1116 Packer, D. L.: Evolution of mapping and anatomic imaging of cardiac arrhythmias. J. Cardiovasc. Electrophysiol., 2004, 15 , 839

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Improved survival with an implanted defibrillator in patients with coronary disease at high risk for ventricular arrhythmia N Engl J Med 335 1933 1940

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. Gorelik 2009 Embryonic stem cell-derived cardiomyocytes as a model to study fetal arrhythmia related to maternal disease J Cell Mol Med 13 3730

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Orvosi Hetilap
Authors: Tamás Szili-Török, Szabolcs Szeghy, Attila Kardos, László Környei, Dóra Paprika, András Szatmári, and András Temesvári

Hebe, J., Krings, G., Hansen, P. és mtsai: Arrhythmias in patients with congenital heart disease and their impact on prognosis. Herz, 1999, 24 , 315

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Orvosi Hetilap
Authors: István Lőrincz, Eszter Szánthó, József Simkó, Zoltán Szabó, Kitti Barta, Márta Füzi, and Gyula Szigeti

Rosenbaum, D. S., Jackson, L. E., Smith, J. M. és mtsai: Electrical alternans and vulnerability to ventricular arrhythmias. N. Engl. J. Med., 1994, 330 , 235–241. Smith J. M

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Arrhythmiarizikó és cardialis szövődmények áramütéses balesetet követően

Szakirodalmi áttekintés és sürgősségi ellátási protokoll

Orvosi Hetilap
Authors: Dávid Pilecky, Enikő Kovács, and Endre Zima

injury. [Herzschädigung nach Stromunfall.] Versicherungsmedizin 1991; 43: 148–151. [German] 32 Pilecky D, Vamos M, Bogyi P, et al. Risk of cardiac arrhythmias

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: Respiratory sinus arrhythmia: a frequency dependent phenomenon. J. Appl. Physiol. 19, 479–482 (1964) Coulter N.A. Respiratory sinus arrhythmia: a frequency dependent phenomenon J. Appl. Physiol. 1964 19

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Interventional Medicine and Applied Science
Authors: Zsolt Komka, E. Bosnyák, E. Trájer, A. Protzner, Zs. Major, G. Pavlik, M. Tóth, and A. Udvardy

Abstract

Sudden cardiac death (SCD) of athletes usually occurs during warm-up or shortly after training. At this point sympathetic tone is still elevated but oxygen demand does not differ from resting levels. It is supposed not to have a primarily ischemic origin but most likely relates to repolarization abnormalities which can be associated with intracellular cAMP level caused by increased sympathetic tone. The mediators of sympathetic nervous system are the catecholamines (epinephrin, norepinephrin). Measuring QT-dispersion can show the repolarization's inhomogeneity. 27 elite soccer players, 28 triathletes and 29 non-trained control person took part in our study. It was recorded cardiac ultrasound, an ECG and taken blood before and after exercise. We found significantly higher QT-dispersion and catecholamines in soccer players compared to the triathletes and the controls. However the soccer players did not show larger athlete's heart than the triathletes. After exercise the increased repolarization inhomogeneity persisted in soccer players, but in triathletes it decreased. Increased sympathetic tone in athletes can enhance arrhythmia propensity. Our data may explain why the soccer players die of sudden cardiac death most commonly in Europe.

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