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Acta Veterinaria Hungarica
Authors: Noémi Tarpataki, Kinga Pápa, J. Reiczigel, P. Vajdovich and K. Vörös

Kristensen, F. (2002): Treatment of atopic dermatitis. Dermatology—Vet-Med-Lab 3 , Ludwigsburg. Treatment of atopic dermatitis 3

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Acta Veterinaria Hungarica
Authors: Adriana Bravo-Monsalvo, Juan Vázquez-Chagoyán, Lilia Gutiérrez and Héctor Sumano

50 265 282 DeBoer, D. J. and Hillier, A. (2001): The ACVD task force on canine atopic dermatitis (XV): fundamental concepts in clinical diagnosis

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Abramovits, W. Atopic dermatitis. J. Am. Acad. Dermatol., 2005, 53 , S86–S93. Abramovits W. Atopic dermatitis

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Atopic dermatitis (AD) is a genetically predisposed inflammatory and pruritic allergic skin disease with characteristic clinical features. New results on the pathogenesis and therapeutic aspects are discussed in this review. IgE-mediated hypersensitivity may be involved in the largest subset of atopic patients, yet there is another subset for which such involvement cannot be documented. Alterations in epidermal barrier function, priming of cutaneous antigen-presenting cells with IgE, intrinsic keratinocyte defects, and development of autoimmunity are also factors that contribute to the primary disease. Polymorphisms in regions of the genome that are of key importance to the inflammatory response contribute to the patient's clinical picture. Secondary infections, especially with Staphylococcus and yeast organisms, strongly modify or augment the inflammatory response, which changes over time. After the treatment of secondary infections and skin inflammation the avoidance of causal allergens would prevent relapse. Another causative therapy is the variously effective allergen-specific immunotherapy. The newest treatments for canine AD (cyclosporin A and tacrolimus) are highly effective at suppressing the allergic response and comparable to treatment with glucocorticoids. Canine AD presents a substantial diagnostic and therapeutic challenge over a patient's lifetime, and no single treatment is universally effective.

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Arlian, L. G., Schumann, R. J., Morgan, M. S. and Glass, R. L. (2003): Serum immunoglobulin E against storage mite allergens in dogs with atopic dermatitis. Am. J. Vet. Res. 64 , 32–36. Glass R. L

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References 1 Kim, B. S.: Atopic dermatitis. Medscape. http://emedicine.medscape.com/article/1049085-overview#aw2aab6b2b2 2

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Acta Veterinaria Hungarica
Authors: Torsten Schäfer, Jessica Merkl, Eckart Klemm, Heinz-Erich Wichmann and Johannes Ring

Hillier, A. and Griffin, C. (2001): The ACVD task force on canine atopic dermatitis (I): incidence and prevalence. Vet. Immunol. Immunopathol. 81 , 147–151. Griffin

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(pollen-food allergy syndrome). Dermatitis 2015; 26: 78–88. 12 Pónyai Gy, Kiss D, Németh I, et al. Adverse reactions to foods in adulthood atopic dermatitis

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Orvosi Hetilap
Authors: Ferenc Harangi, Katalin Lőrinczy, Anett Lázár, Mária Örkényi, Mária Adonyi and Béla Sebők

Grize, L., Gassner, M., Wuthrich, B. és mtsai: Trends in prevalence of asthma, allergic rhinitis and atopic dermatitis in 5–7-year-old Swiss children from 1992 to 2001. Allergy, 2006, 61 , 556

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Absztrakt

Az eosinophil oesophagitis a nyelőcső krónikus, antigénmediált gyulladása. A nyelőcsőben orális és/vagy aeroantigének által indukált eosinophilgranulocyta-infiltráció, nyálkahártya-hyperplasia és a subepithelialis rétegek fibrosisa szűkülethez, dysphagiához és falatelakadáshoz vezethet. A betegség gyakran társul más allergiás kórképekkel, mint az asthma vagy az atopiás dermatitis. Az eosinophil oesophagitis kezelésének sarokkövei az elkerülő diéta és a helyileg alkalmazott, gyulladáscsökkentő szteroidkezelés. A szerzők áttekintik a jelenleg rendelkezésre álló kezelési stratégiákat. Orv. Hetil., 2015, 156(23), 927–932.

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