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) Vitamin e protects chondrocytes against hydrogen peroxide-induced oxidative stress in vitro . Inflamm. Res. 62 , 781 – 789 . 4. Bonifacio , A. , Beleites , C. , Vittur , F
. Neill , S. J. , Desikan , R. , Clarke , A. , Hurst , R. , Hancock , J. ( 2002 ) Hydrogen peroxide and nitric oxide as signalling molecules in plants . J. Exp. Bot. 53 , 1237 – 1247 . 3
utilizing the principle of protein-dye binding . Anal. Biochem. 72 : 248 – 254 . Brennan , T. , Frenkel , C. 1977 . Involvement of hydrogen peroxide in the regulation of
References Al-Daoude , A. , Jawhar , M. , Arabi , M.I.E. 2013 . Hydrogen peroxide induction in barley- Cochliobolus sativus interaction . J. Plant Pathol
Abstract
Hydrogen peroxide containing impurities has caused a lot of explosion accidents. In this study, a simple device that using a glass vessel was made, cupric chloride was added into hydrogen peroxide, and properties of runaway reaction of hydrogen peroxide were evaluated. As a result, when copper ion exists over 0.04%, 50 g of 30%-hydrogen peroxide has caused runaway reaction. Besides, it has been confirmed that the shape of the reactor and initial temperature influence runaway reaction.
Abstract
All thermal systems are subject to problems of thermal regulation. These can be understood through the use of thermochemical systems, in particular for those in the liquid phase. A dynamic linear model was earlier applied to obtain both the reaction enthalpy and the rate constant at constant temperature for the catalytic decomposition of hydrogen peroxide. This first model did not yield a good fitting between the calculated and experimental data. The hypothesis that the rate constant was independent of temperature was too strong. In the present study, a more elaborate, non-linear model was developed, which takes into account the rate constant variations as a function of temperature (Arrhenius law). This model allowed the activation energy to be determined. The calculated data then successfully fitted the experimental data. The literature indicates that the first-order rate law is not valid for a certain range of concentrations; the present model verified this. The results of dynamic modelling confirm and increase the precision of results obtained in different ways. The developed model is validated through these comparisons.
Stress sensitivity of three related phytopathogenic Fusarium species (Fusarium graminearum, Fusarium oxysporum and Fusarium verticillioides) to different oxidative, osmotic, cell wall, membrane, fungicide stressors and an antifungal protein (PAF) were studied in vitro. The most prominent and significant differences were found in oxidative stress tolerance: all the three F. graminearum strains showed much higher sensitivity to hydrogen peroxide and, to a lesser extent, to menadione than the other two species. High sensitivity of F. verticillioides strains was also detectable to an azole drug, Ketoconazole. Surprisingly, no or limited differences were observed in response to other oxidative, osmotic and cell wall stressors. These results indicate that fungal oxidative stress response and especially the response to hydrogen peroxide (this compound is involved in a wide range of plant-fungus interactions) might be modified on niche-specific manner in these phylogenetically related Fusarium species depending on their pathogenic strategy. Supporting the increased hydrogen peroxide sensitivity of F. graminearum, genome-wide analysis of stress signal transduction pathways revealed the absence one CatC-type catalase gene in F. graminearum in comparison to the other two species.
. , Bhattacharjee , S. , Armin , S.-M. , Qian , P. , Xin , W. , Li , H.-Y. , Burritt , D. J. , Fujita , M. , Tran , L.-SP. ( 2015 ) Hydrogen peroxide priming modulates abiotic oxidative stress tolerance: insights from ROS detoxification and
O’Connor, L. F, Scmid-Schonbein, G. W.: Plasma hydrogen peroxide production in hypertension subjects at genetic risk of hypertension. J. Hypertension, 1998, 16 , 291–303. Scmid-Schonbein G. W
Infection of some leaves of Xanthi-nc tobacco with tobacco mosaic virus (TMV) induces systemic acquired resistance (SAR) in remote leaves of the plant to a second (challenge) infection, and therefore produces only a limited necrotization in the resistant leaves. Here we show that the levels of superoxide and hydrogen peroxide are lower in the remote infected leaves exhibiting the SAR. Treatment of leaves of Xanthi-nc tobacco with benzothiadiazole (BTH) also suppresses tissue necrotization and accumulation of superoxide and hydrogen peroxide upon TMV inoculation. However, both of these reactive oxygen species are up-regulated and tissue necrotization is increased in a transgenic NahG tobacco, which is unable to produce a SAR response. Treatment of TMV-infected NahG leaves with BTH also resulted in a reduced level of necrotization and an attenuated accumulation of superoxide and hydrogen peroxide after inoculation with TMV. Thus, the level of reactive oxygen species seems to be correlated with the size and number of necrotic lesions caused by TMV. It would seem that reactive oxygen species play a pivotal role in TMV-induced cell death response.
