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Orvosi Hetilap
Authors: Enikő Kovács, Zsigmond Jenei, Anikó Horváth, László Gellér, Szabolcs Szilágyi, Ákos Király, Levente Molnár, Péter Sótonyi jr., Béla Merkely and Endre Zima

Sinclair, H. R., Andrews, P. J.: Bench-to-bedside review: Hypothermia in traumatic brain injury. Crit. Care, 2010, 14 , 204. Andrews P. J

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Introduction Hypothermia has been shown to induce significant changes in responsiveness of vascular smooth muscle cells to various drugs ( 6 , 7 ). Most of the previous studies examining the effect of hypothermia on smooth

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Gunn, A. J.: Cerebral hypothermia for prevention of brain injury following perinatal asphyxia. Curr. Opin. Pediatr., 2000, 12(2) , 111–115. Gunn A. J

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Orvosi Hetilap
Authors: Kata Kovács, Enikő Szakmár, Ünőke Méder, Márton Kolossváry, Zsolt Bagyura, Lilla Lamboy, Zsuzsanna Élő, Attila Szabó, Miklós Szabó and Ágnes Jermendy

–291. 4 Azzopardi, D., Strohm, B., Marlow, N., et al.: Effects of hypothermia for perinatal asphyxia on childhood outcomes. N. Engl. J. Med., 2014, 371 (2), 140

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Introduction Temperature can influence endothelial and vascular smooth muscle cell function and alter the reactivity of the vessels. Moderate hypothermia, cooling to 25–31 °C, has been shown to have a variable influence on the

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Orvosi Hetilap
Authors: Vince Pongor, Gergely Toldi, Miklós Szabó and Barna Vásárhelyi

, physiological effects, and complications of hypothermia. Crit. Care Med., 2009, 37 , S186–S202. Polderman K. H. Mechanisms of action, physiological effects, and complications of hypothermia

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Orvosi Hetilap
Authors: Dávid Pilecky, Gábor Szudi, Enikő Kovács, Zsigmond Jenei, László Gellér, Krisztina Heltai, Levente Molnár, György Bárczi, Dávid Becker, Béla Merkely and Endre Zima

References 1 Hypothermia after Cardiac Arrest Study Group: Mild therapeutic hypothermia to improve the neurologic outcome after cardiac arrest. N. Engl. J

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prediction after cardiac arrest. Clin Neurophysiol Pract. 2017; 2: 201–205. 8 Hypothermia after Cardiac Arrest Study Group. Mild therapeutic hypothermia to improve the neurologic

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Heat shock proteins are chaperones that play a pivotal role in controling multiple regulatory pathways such as stress defense, hormone signaling, cell cycle control, cell proliferation and differentiation, and apoptosis. In this study, the expression patterns of four well-known heat shock genes (hsp70, hsc70-1, hsc70-2 and hsp90α) were characterized in the skin, spleen and blood cells of the common carp, under unstressed conditions and after Cd2+ treatment or hypothermia. The examined genes were expressed in a tissue-specific manner: hsc70-2 was expressed constitutively, and was at best only slightly inducible; hsp90α exhibited a high basic expression in all three tissues, whereas hsc70-1 did so only in the blood cells, the expression of hsp70 proved to be below the level of detection in unstressed fish. Cold shock induced the expression of hsp genes in the spleen (hsp90α) and blood cells (hsp70, hsc70-1 and hsp90α), while Cd2+ treatment has no effect on the expression pattern. The highest inducibilities were detected in the skin: for hsp70 an induction of at least 20-fold after cadmium exposure, for hsc70-1 of at least 30-fold and for hsp90α of 3-fold after hypothermia.

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hypothermia. Anesthesiology, 2001, 95 , 531–543. Sessler D. I. Complications and treatment of mild hypothermia Anesthesiology

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