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, are protected from stable C. jejuni colonization even following peroral infection with high bacterial loads [ 17 ]. This physiological colonization resistance provided by the intact complex murine gut microbiota is abrogated upon broad

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. Conventional laboratory mice, for instance, are protected from C. jejuni infection even after peroral challenge with high bacterial loads due to the distinct complex murine gut microbiota composition providing an effective colonization resistance to the host

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(i.e., secondary abiotic mice) following broad-spectrum antibiotic treatment were unaffected from high-dose T. gondii infection, secondary abiotic mice with a reconstituted murine gut microbiota following fecal microbiota transplantation (FMT

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Medizin (Charité – University Medicine, Berlin, Germany). The murine gut microbiota was depleted as described previously [ 27 ]. In brief, 8-weeks-old mice were transferred into sterile cages and subjected to a broad-spectrum antibiotic treatment for 8 to

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European Journal of Microbiology and Immunology
Authors: Manja Boehm, Daniel Simson, Ulrike Escher, Anna-Maria Schmidt, Stefan Bereswill, Nicole Tegtmeyer, Steffen Backert and Markus M. Heimesaat

revealed that this commensal polymicrobial barrier can be overcome by complete eradication of the murine gut microbiota [ 109 – 112 ]. Remarkably, mice in which the depleted microbiota had been reconstituted with human gut microbiota following faecal

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