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Alving K., Weitzberg E., Lundberg J. M.: Increased amount of nitric oxide in exhaled air of asthmatics. Eur. Respir. J., 1993, 6 , 1368–1370. Lundberg

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An, L., Liu, Y., Zhang, M., Chen, T., Wang, X. 2005. Effects of nitric oxide on growth of maize seedling leaves in the presence or absence of ultraviolet-B radiation. J. of Plant Physiol. 162 :317

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Schuman, E. M., Madison, D. V. (1994) Nitric oxide and synaptic function. Annu. Rev. Neurosci. 17 , 153-183. Nitric oxide and synaptic function Annu. Rev

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Introduction Nitric oxide (NO) is one of the most important molecules that are responsible for the vasodilator tone required for the regulation of blood pressure [ 1 ]. NO is synthesized from L -arginine by a family of enzymes

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calcium into the cells of the heart and blood vessel walls, allowing vascular and other smooth muscle cells to relax and result in lower blood pressure ( 21 ). Nitric oxide is an endogenous endothelium-derived vasodilator. Although efforts are

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. Böhm , F. M. L. Z. , Ferrarese , M. L. L. , Zanardo , D. I. L. , Magalhaes , J. R. , Ferrarese-Filho , O. ( 2010 ) Nitric oxide affecting root growth, lignification and related enzymes in soybean seedlings . Acta Physiol. Plant. 32 , 1039

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Prevot, V., Croix, D., Rialas, C. M., Poulain, P., Fricchione, G. L., Stefano, G. B., Beauvillain, J. C. (1999) Estradiol coupling to endothelial nitric oxide stimulates gonadotropin-releasing hormone release

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. 2003 91 129 141 Beligni, M. V., Lamattina, L. (2000) Nitric oxide stimulates seed germination and de-etiolation, and

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superoxide with nitric oxide to form peroxynitrite in alkaline solution. Inorganic Chem. 24 :3504–3505. Zafiriou D.C. Reaction of superoxide with nitric oxide to form peroxynitrite in

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Acta Physiologica Hungarica
Authors: Z Kojić, Z Kojić, Z Kojić, Lj Šćepanović, Lj Šćepanović, Lj Šćepanović, N Popović, N Popović, and N Popović

In our previous work we have shown that in mouse heart basal level of endothelial produced nitrite, as a marker of nitric oxide (NO) formation, was 9.7 nmol l-1. Bradykinin (10 mmol l-1) induced a 5-fold rise in nitrite release, the coronary venous effluent concentration being 58 nmol l-1, but there was no effect on myocardial oxygen consumption (MVO2). The aim of this study was to assess the levels of authentic nitric oxide solution, exogenously applied, on myocardial oxygen consumption. Isolated mouse hearts (n=36) were paced (500 imp./min) and perfused at constant flow (16.0±0.3 ml g-1 min-1). When coronary vasculature resistance was carefully controlled by adenosine (1 mmol l-1), authentic nitric oxide solution, in a concentration less than 5 mmol l-1 did not alter myocardial oxygen consumption. Only concentrations of nitric oxide higher than 5 mmol l-1 induced reduction in myocardial oxygen consumption. Thus in the saline perfused mouse heart, with carefully controlled vasodilatation, modulating myocardial nitric oxide levels using an arterial application of authentic nitric oxide, concentrations higher than 5 mmol l-1 of nitric oxide were required to induce a decrease in myocardial oxygen consumption.

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