Schulmann, K., Schwiegel, W.: Capsule endoscopy for small bowel surveillance in hereditary intestinal polyposis and non-polyposis syndromes. Gastrointest. Endosc. Clin. N. Am., 2004, 14 , 149
. Eur. Arch. Otorhinolaryngol., 2008 (in press). Corradini, C., Del Ninno, M., Buonomo, A. és mtsai: Amphotericin B and lysine acetylsalicylate in the combined treatment of nasal polyposis associated
63 Graham, J. R.: Gastric polyposis: onset during long-term therapy with omeprazole. Med. J. Aust., 1992, 157 , 287–288. Graham J. R
750 Jarvinen, H. J. Genetic testing for polyposis: practical and ethical aspects. Gut, 2003, 52 , 19–22. Jarvinen H. J
Absztrakt
A szerzők egy 48 éves férfi kórtörténetét ismertetik. Az eset diagnosztikus különlegessége hogy a colonoscopia során számos barnás színű, egy-két cm nagyságú sigmapolipot és diverticulosist találtunk. A gastrointestinalis vérzés miatt laparoscopos sigmaresectiót ajánlottunk, melyet tervezett időben elvégeztünk. A hisztológiai vizsgálat a sigmabélben a diverticulosis és a polyposis együttes előfordulását megerősítette. A ritka elváltozás az irodalomban diverticulosisasszociált, prolapsus típusú gyulladásos polyposisként ismert, ami a mucosalis prolapsus egy változata. A polipok barnás elszíneződését a bennük lerakódott haemosiderin okozta.
The fungal revolution taking place in otorhinology inspired us to study the frequency of occurrence of fungi in the nasal mucus of chronic rhinosinusitis (CRS) patients (with or without polyposis) in order to evaluate the incidence of eosinophilic fungal sinusitis in CRS patients. Ninety-six samples were examined from patients with CRS. In 74 cases mucus was collected non-invasively, and in 22 cases during operation. The Gram-stained direct smears of all samples were also evaluated. Bacteria and fungi colonizing in the mucus were detected by culturing method. The control group consisted of 50 healthy volunteers. Typical aerobic pathogenic bacteria could be isolated from 34 patients. Fifty-seven aerobic bacteria were isolated, i.e. 1.6 bacteria/positive patient with a maximum of 3 different bacteria/sample. The most frequently isolated bacteria were Staphylococcus aureus, Escherichia coli, Klebsiella pneumoniae, Streptococcus pneumoniae, and Haemophilus influenzae. Yeasts and moulds could be detected from 79 patients (83%): Candida albicans, Candida spp., Aspergillus spp., Cladosporium spp, and Penicillium spp. were isolated most frequently. Altogether 237 yeasts and moulds were isolated, i.e. 3.0 different fungi/positive patient, with a maximum of 5 different fungi/sample. In the control group aerobic pathogens were not isolated, only apathogenic species. Fungi were isolated from 22 healthy patients (44%). These data indicate that fungi are frequently involved in the aetiology of CRS. IgE-mediated hypersensitivity to fungal allergens could not be proven in our patients.
JA Morgan D Silverberg RM Steinhagen 2004 Familial polyposis coli: clinical manifestations
intestinal polyposis, a predisposing factor for carcinoma of colon and rectum. Am J Hum Genet 1951; 3: 167–168. 7 Gardner EJ, Plenk HP: Hereditary pattern for multiple
D Couturier B Bressac-de_Paillerets 2000 Extensive molecular screening for non-polyposis colorectal cancer British J of
] 10 Ward EM, Wolfsen HC. Review article: the non-inherited gastrointestinal polyposis syndromes. Aliment Pharmacol Ther. 2002; 16: 333–342. 11
