Search Results

You are looking at 1 - 10 of 753 items for :

  • Refine by Access: All Content x
Clear All

., Agnel, J. P., Tronchet, M., Ponchet, M., Blein, J. P., Roby, D., Triantaphylides, C. and Montillet, J. L. (2005): The combined action of 9-lipoxygenase and galactolipase is sufficient to bring about programmed cell death during tobacco hypersensitive

Restricted access

. Air Pollution and Plant Life 2004 Borowiak, K. (2005) Visible leaf injury of tobacco plants caused by tropospheric ozone in the

Restricted access

A. Rodgman and T. A. Perfetti, The chemical components of tobacco and tobacco smoke, Taylor & Francis, Boca Raton, FL, 2008, pp. 780–789 Perfetti T. A

Full access

Tobacco Told in Sixteenth-Century Europe. Environmental History Vol. 9, No. 4. (accessed 13 Feb. 2005), pp. 648–678 Mancall P. C

Restricted access

Allan, A. C., Lapidot, M., Culver, J. N. and Fluhr, R. (2001): An early tobacco mosaic virus-induced oxidative burst in tobacco indicates extracellular perception of the virus coat protein. Plant Physiol. 126, 97–108. Fluhr R

Restricted access

Chatzivassiliou, E. K., Boubourakas, I., Drossses, E., Eleftherohorinos, I., Jenser, G., Peters, D. and Katis, N. I. (2001): Weeds in greenhouses and tobacco fields are differentially infected by tomato spotted

Restricted access

An efficient and sensitive analytical method based on precolumn derivatization and gas chromatography—mass spectrometry—selected ion monitoring (GC—MS—SIM) was proposed and validated for analysis of two cembrenediols (CBDs) which are α-cembrenediol and β-cembrenediol in tobacco samples. CBDs in tobacco samples were extracted by sonication with 50 mL dichloromethane for 10 min before derivatized with 2:3 (v/v) bis(trimethylsilyl)trifluoroacetamide (BSTFA)—pyridine at 20 °C for 100 min. CBDs’ level in tobacco samples was analyzed by GC—MS—SIM and quantified by the internal standard method. The linear range for α-CBD and β-CBD was 13.6–554.6 μg mL−1 and 4.11–162.6 μg mL−1, and the correlation coefficients of both were 0.9998. The limit of detection (LOD) and limit of quantification (LOQ) of α-cembrenediol and β-cembrenediol were 0.40 μg g−1 and 1.34 μg g−1, and 0.27 μg g−1 and 0.90 μg g−1, respectively. Average recoveries of α-CBD and β-CBD were 94.4–99.9% and 91.9–98.2% while the relative standard deviations (RSDs, n = 5) were ranged from 2.67 to 5.6% and 2.04 to 4.22%, respectively. This proposed analytical method has been successfully applied to analyze CBDs in tobacco samples.

Open access



Obsessive-compulsive disorder (OCD) is a biologically heterogeneous neuropsychiatric disorder. It is associated with impulsive as well as compulsive neurocognitive mechanisms. Cigarette smoking is common among most psychiatric patients; however, OCD patients are thought to show reduced rates. OCD smokers may thus represent a relatively uncommon OCD subtype, characterised by increased impulsivity. In this study, we aim to establish the prevalence of smoking in a large, well-defined OCD cohort. We investigate whether smokers with OCD differ from non-smokers with OCD on clinical measures of behavioural impulsivity and domains of personality and temperament, including reward-dependence and novelty-seeking.


183 of 200 outpatients with DSM-IV OCD were interviewed to determine smoking status. A sub-sample of 10 smokers was compared with 10 non-smokers, pair wise matched for age and gender. Patients were assessed for DSM co-morbidity, symptom profile, OCD severity, behavioural impulsivity and personality dimensions.


Only 10 individuals (5.46%; five males) were smokers. Compared to OCD non-smokers, OCD smokers scored significantly higher on the Barratt Impulsiveness Scale (p < 0.001). They also scored significantly higher on TCI measures of novelty seeking (p < 0.001) and reward dependence (p < 0.001) and significantly lower on measures of harm avoidance (p < 0.001).


Tobacco smoking is rare in OCD. Significantly higher levels of behavioural impulsivity and temperamental factors associated with reward driven impulsivity are seen in OCD smokers compared to non-smokers. Tobacco smoking may indicate a possible source of neurocognitive heterogeneity in OCD.

Open access

Clayton, R.A. (1959): Properties of tobacco polyphenol oxidase. Arch. Biochem. Biophys. , 81 , 404–417. Clayton R

Restricted access

979 Ádám, A., Farkas, T., Somlyai, G., Hevesi, M. and Király, Z. (1989): Consequence of O 2 ·− generation during a bacterially induced hypersensitive reaction in tobacco: deterioration of

Restricted access