Authors:
Ji-Bin Li Department of Clinical Research, State Key Laboratory of Oncology in South China, Collaborative Innovation Center for Cancer Medicine, Sun Yat-sen University Cancer Center, Guangzhou, China
Centre for Health Behaviours Research, The Jockey Club School of Public Health and Primary Care, The Chinese University of Hong Kong, Hong Kong, China

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Phoenix K. H. Mo Centre for Health Behaviours Research, The Jockey Club School of Public Health and Primary Care, The Chinese University of Hong Kong, Hong Kong, China
Shenzhen Research Institute, The Chinese University of Hong Kong, Shenzhen, China

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Joseph T. F. Lau Centre for Health Behaviours Research, The Jockey Club School of Public Health and Primary Care, The Chinese University of Hong Kong, Hong Kong, China
Shenzhen Research Institute, The Chinese University of Hong Kong, Shenzhen, China

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Xue-Fen Su Centre for Health Behaviours Research, The Jockey Club School of Public Health and Primary Care, The Chinese University of Hong Kong, Hong Kong, China
Shenzhen Research Institute, The Chinese University of Hong Kong, Shenzhen, China

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Xi Zhang Clinical Research Unit, Xin Hua Hospital – Shanghai Jiao Tong University School of Medicine, Shanghai, China

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Anise M. S. Wu Faculty of Social Sciences, Department of Psychology, University of Macau, Macao, China

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Jin-Cheng Mai Department of Psychological Health Research, Center for Health Promotion of Primary and Secondary School of Guangzhou, Guangzhou, China

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Yu-Xia Chen Department of Psychological Health Research, Center for Health Promotion of Primary and Secondary School of Guangzhou, Guangzhou, China

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Open access

Background and aims

The aim of this study is to estimate the longitudinal associations between online social networking addiction (OSNA) and depression, whether OSNA predicts development of depression, and reversely, whether depression predicts development of OSNA.

Methods

A total of 5,365 students from nine secondary schools in Guangzhou, Southern China were surveyed at baseline in March 2014, and followed up 9 months later. Level of OSNA and depression were measured using the validated OSNA scale and CES-D, respectively. Multilevel logistic regression models were applied to estimate the longitudinal associations between OSNA and depression.

Results

Adolescents who were depressed but free of OSNA at baseline had 1.48 times more likely to develop OSNA at follow-up compared with those non-depressed at baseline [adjusted OR (AOR): 1.48, 95% confidence interval (CI): 1.14–1.93]. In addition, compared with those who were not depressed during the follow-up period, adolescents who were persistently depressed or emerging depressed during the follow-up period had increased risk of developing OSNA at follow-up (AOR: 3.45, 95% CI: 2.51–4.75 for persistent depression; AOR: 4.47, 95% CI: 3.33–5.99 for emerging depression). Reversely, among those without depression at baseline, adolescents who were classified as persistent OSNA or emerging OSNA had higher risk of developing depression compared with those who were no OSNA (AOR: 1.65, 95% CI: 1.01–2.69 for persistent OSNA; AOR: 4.29; 95% CI: 3.17–5.81 for emerging OSNA).

Conclusion

The findings indicate a bidirectional association between OSNA and depression, meaning that addictive online social networking use is accompanied by increased level of depressive symptoms.

Abstract

Background and aims

The aim of this study is to estimate the longitudinal associations between online social networking addiction (OSNA) and depression, whether OSNA predicts development of depression, and reversely, whether depression predicts development of OSNA.

Methods

A total of 5,365 students from nine secondary schools in Guangzhou, Southern China were surveyed at baseline in March 2014, and followed up 9 months later. Level of OSNA and depression were measured using the validated OSNA scale and CES-D, respectively. Multilevel logistic regression models were applied to estimate the longitudinal associations between OSNA and depression.

Results

Adolescents who were depressed but free of OSNA at baseline had 1.48 times more likely to develop OSNA at follow-up compared with those non-depressed at baseline [adjusted OR (AOR): 1.48, 95% confidence interval (CI): 1.14–1.93]. In addition, compared with those who were not depressed during the follow-up period, adolescents who were persistently depressed or emerging depressed during the follow-up period had increased risk of developing OSNA at follow-up (AOR: 3.45, 95% CI: 2.51–4.75 for persistent depression; AOR: 4.47, 95% CI: 3.33–5.99 for emerging depression). Reversely, among those without depression at baseline, adolescents who were classified as persistent OSNA or emerging OSNA had higher risk of developing depression compared with those who were no OSNA (AOR: 1.65, 95% CI: 1.01–2.69 for persistent OSNA; AOR: 4.29; 95% CI: 3.17–5.81 for emerging OSNA).

Conclusion

The findings indicate a bidirectional association between OSNA and depression, meaning that addictive online social networking use is accompanied by increased level of depressive symptoms.

Introduction

Depression, the most widely reported psychiatric disorder (Knopf, Park, & Mulye, 2008; Thapar, Collishaw, Potter, & Thapar, 2010), is an important public health issue among adolescents. Over 9% of adolescents reported moderate to severe levels of depression, and its 1-year incidence rate was estimated at 3% in the United States (Rushton, Forcier, & Schectman, 2002). In Southern China, our previous study reported a 1-week depression prevalence of 23.5% among secondary school students (Li et al., 2017).

A positive association between Internet addiction and depression among adolescents has been reported in both cross-sectional (Moreno, Jelenchick, & Breland, 2015; Yoo, Cho, & Cha, 2014) and longitudinal studies (Cho, Sung, Shin, Lim, & Shin, 2013; Ko, Yen, Chen, Yeh, & Yen, 2009; Lam, 2014). However, these studies assessed Internet addiction in general rather than specific types of online activities. Adolescents could conduct multiple types of online activities on the Internet. Several studies have highlighted the significance and necessity for distinguishing addiction to specific Internet-related activities from Internet addiction in general (Davis, 2001; Laconi, Tricard, & Chabrol, 2015; Pontes, Szabo, & Griffiths, 2015). Online social networking is a relatively new phenomenon, and high prevalence of depression has been observed among the population who are online social networking users (Lin et al., 2016; Tang & Koh, 2017). Compared to the general population, teenagers and students are the most frequent users of online social networking (Griths, Kuss, & Demetrovics, 2014). Online social networking addiction (OSNA) is a relatively new addictive behavior among adolescents along with compulsive involvement in online social networking activities. As a specific type of Internet-related behavioral addictions, OSNA incorporates core classic symptoms of addiction (Griffiths, 2013; Kuss & Griffiths, 2011), and is defined as “being overly concerned about online social networking use, to be driven by a strong motivation to log on to or use online social networking that impairs other social activities, studies/jobs, interpersonal relationships, and/or psychological health and well-being” (Andreassen, 2015). OSNA has risen noticeably among adolescents. Around 9.78% of the US college students self-perceived to have Facebook addiction (Pempek, Yermolayeva, & Calvert, 2009), and 29.5% of Singaporean college students possess OSNA (Tang & Koh, 2017). A study in 2010 reported that the OSNA prevalence was even higher than 30% in Chinese college students (Zhou & Leung, 2010). Evidences have suggested that excessive and compulsive online social networking is seldom beneficial, rather having potentially detrimental effects on adolescents’ psychosocial well-being, including emotional, relational, and other health-related outcomes (Andreassen, 2015).

A few of cross-sectional surveys reported a positive association between OSNA and depression among adolescents (Hong, Huang, Lin, & Chiu, 2014; Koc & Gulyagci, 2013). However, due to the inherent limitation of the cross-sectional study design, it is still unclear whether OSNA is a cause or consequence of depression or bidirectional. Online social networking could provide adolescents with social convenience and capital, selective self-disclosure, and potential social support (Ellison, Steinfield, & Lampe, 2007; Steinfield, Ellison, & Lampe, 2008). Individuals who experience psychiatric disorders (i.e., depression and anxiety) might view online social networking as a safe and important virtual community (Gámez-Guadix, 2014), where they could escape from emotional problems experienced in the real world (Andreassen, 2015; Griths et al., 2014), and further lead to potential addictive involvement (Oberst, Wegmann, Stodt, Brand, & Chamarro, 2017). Meanwhile, excessive exposure to virtual community would result in negative emotions (McDougall et al., 2016). Adolescents with maladjustment to their depressive moods may experience more detrimental effects of excessive online social networking (Selfhout, Branje, Delsing, Ter Bogt, & Meeus, 2009). Therefore, a bidirectional association between OSNA and depression is theoretically reasonable. However, to our knowledge, there is no prospective study that focused on exploring the longitudinal relationships between OSNA and depression among adolescents and other populations.

Therefore, we designed a prospective study to comprehensively estimate the longitudinal association between depression and OSNA over time, such as whether OSNA predicts development of depression, and whether depression predicts development of OSNA, by considering changes in OSNA and depression status (e.g., remission from disorder) during a 9-month follow-up period.

Methods

Study design

This prospective cohort study was conducted in Guangzhou, Southern China. The baseline survey was conducted from March to April 2014, and the subsequent follow-up survey was conducted at a 9-month interval, using the same procedure.

Participants and sampling

Participants were recruited using a stratified cluster sampling method. One district/county was conveniently selected from each of three regions (i.e., core, suburb, and outer suburb regions) in Guangzhou, respectively (red dots in Figure 1). Three public secondary schools were then conveniently selected from each selected district/county, and a total of nine schools were thus selected. All the seventh- and the eighth-grade students within the selected schools were voluntarily invited to participate in the study. Anonymous questionnaire was self-administrated by participants in the classroom settings with the absence of any teacher, under the supervision of well-trained research assistants.


            Figure 1.
Figure 1.

The location of the study sites

Citation: Journal of Behavioral Addictions 7, 3; 10.1556/2006.7.2018.69

A total of 5,365 (response rate = 98.04%) students completed the baseline survey. The two questionnaires of the same students were matched using last four digits of home telephone number, last four digits of parents’ mobile phone number, last four digits of participants’ identity card number, participants’ date of birth, last letter of self and parents’ spell name. Finally, 4,871 of 5,365 participants provided complete questionnaires at follow-up (follow-up rate = 90.8%). After excluding those who did not use online social networking (n = 643), a total of 4,237 participants were involved in our longitudinal study.

Measures

Depression

Level of depressive symptoms was measured using the 20-item Chinese version of the Center for Epidemiology Scale for Depression (CES-D). Its psychometric properties have been validated among Chinese adolescents (Chen, Yang, & Li, 2009; Cheng, Yen, Ko, & Yen, 2012; Lee et al., 2008; Wang et al., 2013). Higher scores indicate more severe level of depressive symptoms, with a total score ranging from 0 to 60 (Radloff, 1977). The Cronbach’s α coefficients in this study were .86 at baseline and .87 at follow-up, showing a good internal reliability. Individual reporting a CES-D score ≥21 is defined as a depressed case (Stockings et al., 2015). Following the previous studies (Penninx, Deeg, van Eijk, Beekman, & Guralnik, 2000; Van Gool et al., 2003), change in depression status during follow-up period in this study was categorized as follows: no depression (participants without depression both at baseline and follow-up), remission from depression (participants with depression at baseline but transitioned to without depression at follow-up), persistent depression (participants with depression both at baseline and follow-up), and emerging depression (participants without depression at baseline but transitioned to with depression at follow-up).

Online social networking addiction (OSNA)

Addictive level to online social networking was measured using an OSNA scale, which includes eight items measuring core addictive symptoms of cognitive and behavioral salience, conflict with other activities, euphoria, loss of control, withdrawal, relapse, and reinstatement. Higher scores of OSNA scale indicate higher levels of addictive tendency to online social networking, with a maximum score of 40. Its psychometric properties have been thoroughly assessed in our previous study (Li et al., 2016). There is no established cut-off value for the OSNA scale to identify OSNA cases: participants who scored in the 10th decile of scores (i.e., OSNA score ≥24) were classified as OSNA cases at baseline, and the same cut-off value was used to classify cases at follow-up. The similar classification strategy has been applied in the previous study (Verkuijl et al., 2014). The Cronbach’s α coefficients of OSNA scale in this study were .86 at baseline and .89 at follow-up. Similarly, change in OSNA status from baseline to follow-up was categorized as follows: no OSNA (participants without OSNA both at baseline and follow-up), remission from OSNA (participants with OSNA at baseline but transitioned to without OSNA at follow-up), persistent OSNA (participants with OSNA both at baseline and follow-up), and emerging OSNA (participants without OSNA at baseline but transitioned to with OSNA at follow-up).

Covariates

Covariates included sex, grade, parental education levels, perceived family financial situation, living arrangement (with both parents or not), self-reported academic performance, and perceived study pressure at baseline.

Statistical analyses

Descriptive statistics (e.g., means, standard deviation, and percentages) were presented when appropriate. Intraclass correlation coefficients for clustering across schools were 1.56% (p = .002) for incident depression and 1.42% (p = .042) for incident OSNA, indicating significant variances across schools (Wang, Xie, & Fisher, 2009). Multilevel logistic regression models (Level 1: student; Level 2: school) were therefore applied to evaluate the longitudinal associations between OSNA and depression over time, accounting for the cluster sampling effect from school. Background covariates associated with incident depression/OSNA with p < .05 in univariate analysis or widely reported in the literature (i.e., sex and grade) were adjusted for in the multivariable logistic regression models.

For prediction of OSNA on new incidence of depression among participants who were non-depressed at baseline (n = 3,196), we first estimated the odds ratio (OR) of baseline OSNA, both binary variable (i.e., OSNA or not) and continuous variable (OSNA scale scores), on new incidence of depression after adjusting of significant covariates, and then further adjusting of baseline CES-D scale score (Hinkley et al., 2014). We then estimated the prediction of change in OSNA status over time on new incidence of depression, including a model adjusted of significant covariates and a model additionally adjusted of baseline CES-D scale score.

Reversely, the prediction of depression on new incidence of OSNA among participants without OSNA at baseline (n = 3,657) was estimated in a similar manner to that described above with new incidence of OSNA as outcome and depression as an exposure. The prediction of baseline depression (both continuous and categorical version) on new incidence of OSNA and prediction of change in depression status over time on new incidence of OSNA were estimated, respectively.

Statistical analyses were performed using SAS version 9.4 (SAS Institute, Cary, NC, USA). A two-sided p value <.05 was considered statistically significant.

Ethics

The study procedures were carried out in accordance with the Declaration of Helsinki. School consent and permission for the in-school survey were obtained from school principals before the survey had administered. Verbal consent was obtained from students before their participation. This study and the consent procedure were approved by the Survey and Behavioral Research Ethics Committee of the Chinese University of Hong Kong.

Results

Participants’ characteristics and attrition analysis

Attrition analysis showed that there were no significant differences in terms of parental education levels and self-reported academic performance between adolescents who were involved in the longitudinal analysis (n = 4,237) and who were excluded from longitudinal analysis (n = 1,128). Adolescents, who were involved in the longitudinal sample were more likely to be females, were from the eighth grade, have good family financial situation, lived with both parents, and perceive nil/light study pressure (Table 1).

Table 1.

Attrition analysis and participants’ characteristics in the longitudinal sample

Baseline Participants in the longitudinal sample Participants without depression at baseline Participants without OSNA at baseline
Yes No p * Non-OSNA OSNA p * Non-depressed Depressed p *
Total 5,365 4,237 1,128 2,922 274 2,922 735
Sex
 Male 2,533 (47.2) 2,105 (49.7) 727 (64.4) <.001 1,464 (50.1) 164 (59.8) .002 1,464 (50.1) 309 (42.0) <.001
 Female 2,832 (52.8) 2,132 (50.3) 401 (35.6) 1,458 (49.9) 110 (40.2) 1,458 (49.9) 426 (58.0)
Grade
 Seven 2,592 (48.3) 2,011 (47.5) 581 (51.5) .016 1,418 (48.5) 131 (47.8) .820 1,418 (48.5) 337 (45.9) .194
 Eight 2,773 (51.7) 2,226 (52.5) 547 (48.5) 1,504 (51.5) 143 (52.2) 1,504 (51.5) 398 (54.2)
Father’s education level
 Primary school or below 356 (6.6) 273 (6.4) 83 (7.4) .376 165 (5.7) 21 (7.7) .049 165 (5.7) 61 (8.3) .010
 Junior secondary school 1,816 (33.9) 1,425 (33.6) 391 (34.7) 958 (32.8) 108 (39.4) 958 (32.8) 259 (35.2)
 Senior secondary school 1,646 (30.7) 1,312 (31.0) 334 (29.6) 911 (31.2) 79 (28.8) 911 (31.2) 230 (31.3)
 College or above 1,317 (24.5) 1,053 (24.9) 264 (23.4) 763 (26.1) 54 (6.6) 763 (26.1) 159 (21.6)
 Don’t know 230 (4.3) 174 (4.1) 56 (5.0) 125 (4.3) 12 (4.4) 125 (4.3) 26 (3.5)
Mother’s education level
 Primary school or below 588 (11.0) 445 (10.5) 143 (12.7) .144 267 (9.1) 35 (12.8) .108 267 (9.1) 103 (14.0) <.001
 Junior secondary school 1,909 (35.6) 1,507 (35.6) 402 (35.6) 1,030 (35.3) 108 (39.4) 1,030 (35.3) 274 (37.3)
 Senior secondary school 1,497 (27.9) 1,199 (28.3) 298 (26.4) 860 (29.4) 71 (25.9) 860 (29.4) 180 (24.5)
 College or above 1,143 (21.3) 913 (21.6) 230 (20.4) 634 (21.7) 50 (18.3) 634 (21.7) 156 (21.2)
 Don’t know 228 (4.3) 173 (4.1) 55 (4.9) 131 (4.5) 10 (3.6) 131 (4.5) 22 (3.0)
Family financial situation
 Very good/good 2,519 (47.0) 2,047 (48.3) 472 (41.8) <.001 1,495 (51.2) 123 (44.9) .115 1,495 (51.2) 300 (40.8) <.001
 Average 2,664 (49.6) 2,072 (48.9) 592 (52.5) 1,366 (46.7) 143 (52.2) 1,366 (46.8) 405 (55.1)
 Poor/very poor 182 (3.4) 118 (2.8) 64 (5.7) 61 (2.1) 8 (8.6) 61 (2.1) 30 (4.1)
Lives with both parents
 No 4,712 (87.8) 490 (11.6) 163 (14.4) .008 312 (10.7) 30 (11.0) .890 312 (10.7) 107 (14.6) .003
 Yes 653 (12.2) 3,747 (88.4) 965 (85.6) 2,610 (89.3) 244 (89.0) 2,610 (89.3) 628 (85.4)
Academic performance
 Upper 1,817 (33.9) 1,465 (34.6) 223 (19.8) .276 1,142 (39.1) 51 (18.6) <.001 1,142 (39.1) 205 (27.9) <.001
 Medium 2,396 (44.6) 1,920 (45.3) 619 (54.9) 1,306 (44.7) 134 (48.9) 1,306 (44.7) 347 (47.2)
 Lower 1,152 (21.5) 490 (20.1) 286 (25.4) 474 (16.2) 89 (32.5) 474 (16.2) 183 (24.9)
Perceived study pressure
 Nil/light 1,034 (19.3) 811 (19.1) 352 (31.2) <.001 667 (22.8) 31 (11.3) <.001 667 (22.8) 78 (10.6) <.001
 General 3,052 (56.9) 2,433 (57.4) 476 (42.2) 1,769 (60.5) 172 (62.8) 1,769 (60.5) 359 (48.8)
 Heavy/very heavy 1,279 (23.8) 993 (23.4) 300 (26.6) 486 (16.6) 71 (25.9) 486 (16.6) 298 (40.5)

Note. Data are shown as n (%). OSNA: online social networking addiction; CES-D: Center for Epidemiology Scale for Depression; –: not applicable.

p values were obtained using χ2 test.

Among 4,237 adolescents (mean age: 13.9, standard deviation: 0.7) in the longitudinal sample, 49.7% (2,105 of 4,237) were female and 47.5% (2,011 of 4,237) were the seventh grade students. Most of adolescents (88.4%; 3,747 of 4,237) were living with their parents. In the longitudinal sample, the prevalence of depression significantly increased from 24.6% (1,041 of 4,237) at baseline to 26.6% at follow-up (McNemar’s test = 7.459, p = .006). There was no significant difference for the prevalence of OSNA between baseline and follow-up (13.7% at baseline vs. 13.6% at follow-up; McNemar’s test = 0.053, p = .818). A total of 3,196 students were non-depressed at baseline, and 3,657 students were free of OSNA at baseline (Table 1).

Potential confounders associated with new incidence of depression or OSNA

Table 2 shows that perceived poor family financial situation, self-reported poor academic performance, and perceived heavy study pressure were significantly associated with both higher incidence of depression (range of univariate OR: 1.32–1.98) and higher incidence of OSNA (range of univariate OR: 1.61–2.76). Living with their parents was a significantly protective factor for incidence of OSNA only [univariate OR: 0.65, 95% confidence interval (CI): 0.48–0.89].

Table 2.

Univariate associations between background covariates and incidence of depression/OSNA

Incidence of depression Incidence of OSNA
n (%) (n = 515) ORu (95% CI) p n (%) (n = 335) ORu (95% CI) p
Sex
 Male 249 (15.9) 1 168 (8.9) 1
 Female 266 (16.3) 0.96 (0.79, 1.16) .641 167 (9.4) 0.94 (0.75, 1.17) .573
Grade
 Seven 250 (16.1) 1 160 (9.1) 1
 Eight 265 (16.1) 1.00 (0.83, 1.21) .977 175 (9.2) 1.00 (0.80, 1.26) .977
Father’s education level
 Primary school or below 32 (17.2) 1 26 (11.5) 1
 Secondary middle school 190 (17.8) 1.04 (0.69, 1.59) .827 116 (9.5) 0.81 (0.52, 1.28) .377
 High middle school 139 (14.0) 0.80 (0.52, 1.23) .317 93 (8.2) 0.67 (0.42, 1.07) .090
 University or above 129 (15.8) 0.92 (0.60, 1.42) .705 86 (9.3) 0.78 (0.49, 1.26) .310
 Don’t know 25 (18.3) 1.14 (0.63, 2.04) .666 14 (9.3) 0.79 (0.40, 1.59) .516
Mother’s education level
 Primary school or below 47 (15.6) 1 31 (8.4) 1
 Secondary middle school 196 (17.2) 1.15 (0.81, 1.63) .424 118 (9.1) 1.11 (0.73, 1.69) .621
 High middle school 141 (15.2) 1.01 (0.70, 1.46) .939 109 (10.5) 1.28 (0.84, 1.96) .257
 University or above 105 (15.4) 1.03 (0.70, 1.52) .861 64 (8.1) 0.97 (0.61, 1.53) .891
 Don’t know 26 (18.4) 1.32 (0.77, 2.25) .310 13 (8.5) 1.03 (0.52, 2.03) .940
Family financial situation
 Very good/good 229 (14.2) 1 145 (8.1) 1
 Average 269 (17.8) 1.32 (1.08, 1.60) .006 172 (9.7) 1.21 (0.96, 1.53) .105
 Poor/very poor 17 (24.6) 1.98 (1.12, 3.49) .019 18 (19.8) 2.76 (1.60, 4.76) <.001
Lives with both parents
 No 64 (18.7) 1 54 (12.9) 1
 Yes 451 (15.8) 0.80 (0.60, 1.07) .135 281 (8.7) 0.65 (0.48, 0.89) .008
Academic performance
 Upper 169 (14.2) 1 109 (8.1) 1
 Medium 226 (15.7) 1.13 (0.91, 1.41) .254 145 (8.8) 1.10 (0.85, 1.42) .488
 Lower 120 (21.3) 1.66 (1.28, 2.16) <.001 81 (12.3) 1.61 (1.19, 2.19) .002
Perceived study pressure
 Nil/light 96 (13.8) 1 59 (7.9) 1
 Average 305 (15.7) 1.16 (0.90, 1.48) .253 178 (8.4) 1.05 (0.77, 1.44) .735
 Heavy/very heavy 114 (20.5) 1.63 (1.20, 2.20) .002 96 (12.5) 1.65 (1.17, 2.32) .004

Note. OSNA: online social networking addiction; ORu: univariate odds ratio; 95% CI: 95% confidence interval, obtained by the univariate logistic regression models.

OSNA predict new incidence of depression

Among 3,196 adolescents who were non-depressed at baseline, univariate model showed that baseline OSNA was significantly associated with higher incidence of depression during the follow-up period (univariate OR: 1.65, 95% CI: 1.22–2.22). After adjustment of sex, grade, family financial situation, academic performance, and perceived study pressure, the association remained significant [adjusted OR (AOR): 1.48, 95% CI: 1.09–2.01]. When further adjusting of baseline CES-D score, the association becomes statistically non-significant (AOR: 1.16, 95% CI: 0.85–1.60). The similar results were observed when using OSNA score (continuous variable) as a predictor of new incident depression (Table 3).

Table 3.

Longitudinal associations between OSNA and depression: multilevel logistic regression models

n No. of new incident cases Univariate models Multivariable models
ORu (95% CI) p AOR (95% CI) p AOR (95% CI) p
OSNA predict new incident depression (n = 3,196)
Baseline OSNA score (continuous) 1.05 (1.03, 1.07) <.001 1.04 (1.02, 1.06)a <.001 1.01 (0.99, 1.03)b .242
Baseline OSNA
 No 2,922 451 1 1a 1b
 Yes 274 64 1.65 (1.22, 2.22) .001 1.48 (1.09, 2.01) .012 1.16 (0.85, 1.60) .342
Change in OSNA status over time
 No OSNA 2,694 354 1 1a 1b
 Remission from OSNA 179 38 1.77 (1.21, 2.58) .003 1.61 (1.10, 2.37) .015 1.29 (0.87, 1.91) .202
 Persistent OSNA 95 26 2.46 (1.54, 3.93) <.001 2.23 (1.39, 3.58) <.001 1.65 (1.01, 2.69) .044
 Emerging OSNA 228 97 4.89 (3.67, 6.52) <.001 4.67 (3.49, 6.24) <.001 4.29 (3.17, 5.81) <.001
Depression predict new incident OSNA (n = 3,657)
Baseline CES-D score (continuous) 1.05 (1.03, 1.06) <.001 1.04 (1.03, 1.05)c <.001 1.03 (1.01, 1.04)d <.001
Baseline depression
 No 2,922 228 1 1c 1d
 Yes 735 107 2.02 (1.58, 2.58) <.001 1.78 (1.38, 2.31) <.001 1.48 (1.14, 1.93) .004
Change in depression status over time
 No depression 2,471 131 1 1c 1d
 Remission from depression 315 21 1.28 (0.80, 2.07) .307 1.19 (0.73, 1.93) .486 0.97 (0.60, 1.59) .918
 Persistent depression 420 86 4.62 (3.43, 6.21) <.001 4.17 (3.05, 5.69) <.001 3.45 (2.51, 4.75) <.001
 Emerging depression 451 97 4.88 (3.67, 6.50) <.001 4.70 (3.53, 6.28) <.001 4.47 (3.33, 5.99) <.001

Note. OSNA: online social networking addiction; CES-D: Center for Epidemiology Scale for Depression; ORu: univariable odds ratio; AOR: adjusted odds ratio; 95% CI: 95% confidence interval.

aModels were adjusted for sex, grade, family financial situation, academic performance, and perceived study pressure. bModels were adjusted for sex, grade, family financial situation, academic performance, perceived study pressure, and baseline CES-D scale score (continuous variable). cModels were adjusted for sex, grade, family financial situation, living arrangement with parents, academic performance, and perceived study pressure. dModels were adjusted for sex, grade, family financial situation, living arrangement with parents, academic performance, perceived study pressure, and baseline OSNA scale score (continuous variable).

We found a significant association between change in OSNA status and higher incidence of depression. Compared with adolescents who were classified as no OSNA, the risk of developing depression was 1.65 times (95% CI: 1.01–2.69) higher among those with persistent OSNA, and 4.29 times (95% CI: 3.17–5.81) higher among those with emerging OSNA, after adjustment of sex, grade, family financial situation, academic performance, perceived study pressure, and baseline CES-D scores (Table 3).

Depression predict new incidence of OSNA

Among 3,657 adolescents who were free of OSNA at baseline, univariate results demonstrated a significant positive association between baseline depression and higher incidence of OSNA (univariate OR: 2.02, 95% CI: 1.58–2.58). After adjusting of sex, grade, family financial situation, living arrangement with parents, academic performance, and perceived study pressure, the association slightly attenuated but remained significant (AOR: 1.78, 95% CI: 1.38–2.31). The association between baseline depression status and incidence of OSNA was still statistically significant when further adjustment of baseline OSNA scores (AOR: 1.48, 95% CI: 1.14–1.93). The results were still significant when using CES-D score (continuous variable) as a predictor of new incident OSNA (Table 3).

A significant association between change in depression status and incidence of OSNA was observed in multivariable analysis. After adjusting of sex, grade, family financial situation, living arrangement with parents, academic performance, perceived study pressure, and baseline OSNA score, as compared to adolescents without depression, the odds of developing OSNA was 3.45 times (95% CI: 2.51–4.75) higher among those who were persistently depressed, and 4.47 times (95% CI: 3.33–5.99) higher among those who were emerging depressed (Table 3).

Discussion

In this large-scale longitudinal study, we found that adolescents who were depressed but free of ONSA at baseline had a 48% higher risk of developing OSNA within 9-month follow-up period compared with those without depression at baseline, but the prediction of baseline OSNA on new incidence of depression was not supported in this study. Moreover, when the effects of changes in status over time (i.e., remission from depression/OSNA at baseline to non-depression/non-OSNA at follow-up) were considered in the models, the results revealed a bidirectional association between OSNA and depression. Adolescents who were persistently depressed or emerging depressed had a higher risk of developing OSNA compared with those who were no depressed during the 9-month follow-up period. Reversely, adolescents who were persistent OSNA or emerging OSNA also have an increased risk of developing depression compared with those who were no OSNA at both baseline and follow-up.

The difference in results obtained using baseline measures (i.e., baseline OSNA) and changes in status (i.e., change in OSNA status) to predict an incidence outcome (i.e., new incidence of depression) could be explained by the high remission rates from OSNA and depression during the follow-up period. The high natural remission rate of Internet addictive behaviors (49.5%–51.5%) has been observed in two previous longitudinal studies in Taiwan (Ko, Yen, Yen, Lin, & Yang, 2007; Ko et al., 2015). The results from our previous survey in Hong Kong also consistently observed a high incidence of remission from Internet addiction behavior during a 12-month period (59.29 per 100 person-years; Lau, Wu, Gross, Cheng, & Lau, 2017). Similarly, in this study, a large proportion of remission cases from depression (41.4%) and OSNA (58.8%) were observed during the study period. These results indicated that OSNA and depression status in baseline assessment could not be treated as unchangeable conditions over time and hence ignoring the remission effect over time would potentially underestimate the effect of OSNA on depression. Thus, we speculated that the modeling approach involving dynamic changes in OSNA and depression status over time could provide more convincing and robust estimation by ruling out the potential offset effects from remission cases.

The findings in this study suggest a bidirectional association between OSNA and depression among adolescents, indicating that depression renders an individual vulnerability to develop OSNA, and in turn, the negative consequence of OSNA further exacerbates the symptoms of depression. Maladaptive cognitions (i.e., rumination, self-doubt, low self-efficacy, and negative self-appraisal) and dysfunctional behaviors (i.e., using Internet to escape from emotional problems) are critical in the development of Internet-related addictive behaviors (Davis, 2001). Depressed individuals usually present cognitive symptoms and possess positive expectancies for their Internet use that Internet could distract them from negative moods and personal problems (e.g., depression and loneliness; Brand, Laier, & Young, 2014; Wu, Cheung, Ku, & Hung, 2013). In particular, online social networking is attractive to people with mood problems because of its anonymity and absence of social cues (i.e., facial expression, voice inflection, and eye contact) compared to face-to-face communications (Young & Rogers, 1998). Depressed individuals might prefer online social networking as a more secure and less threatening means of communication, as well as a means for regulating their negative moods (i.e., alleviating negative emotions, anxiety, and personal problems). These maladaptive cognition and avoidance coping strategies accelerate the development of OSNA. Excessive online social networking involvement displaces the time spent with family and peers in the real world, and causes withdrawal from interpersonal offline activities, which intensifies the negative moods (e.g., depressive symptoms and loneliness; Kraut et al., 1998), thereby presenting a reciprocal relation.

The findings in this study entail several implications in designing prevention and intervention programs. First, the positive prediction of baseline depression on new incidence of OSNA implies that depressed adolescents are at high risk of developing OSNA later. Intervention strategies of reducing depressive symptoms, that is, reducing maladaptive belief of positive outcome expectancies of Internet use, training social skills, and planning offline leisure activities (Chou et al., 2015), might effectively prevent the development of OSNA. Second, it is meaningful to assess the levels of depressive symptoms as a marker of the vulnerability for OSNA. Interventions and preventions targeting adolescents at high risk with identified depressive symptoms might reduce the odds of experiencing OSNA among school adolescents. Third, for the strong prediction of change in OSNA status (i.e., persistent OSNA and emerging OSNA) on incidence of depression and the prediction of change in depression status (i.e., persistent depression and emerging depression) on incidence of OSNA, it implies that OSNA is highly comorbid with depression, indicating a negative reinforcement mechanism.

There are some implications for future research. First, our results along with previous studies indicated that the level of OSNA and depressive symptoms are dynamic and reversible during the study period rather than random fluctuation in chance (Lau et al., 2017). Future studies involving measures of depression or OSNA are suggested to measure these disorders repeatedly rather than just one time point by assuming them unchangeable over time. In addition, the statistical methodology should consider such status change in modeling specifications, such as using change in pathological status over time rather than baseline status as a predictor of mental health outcomes. Second, it raised a concern whether these disorders (i.e., depressive symptoms and Internet-related behaviors) are long-lasting or short-term. Further longitudinal studies involving latent-class trajectory modeling approach are alternative to estimate the natural developmental course of these disorders.

To our knowledge, our cohort study is the first to estimate a bidirectional association between OSNA and depression among the adolescents. The main strength of this study is a prospective large-scale study design with repeated measures for OSNA and depression. Another major advantage is that a bidirectional association, including the longitudinal prediction of OSNA on development of depression and the longitudinal prediction of depression on development of OSNA, was tested in the same sample.

However, several limitations should be noted when interpreting the findings. First, due to self-reported data collection method, reporting bias may consequentially exist (e.g., social desirable bias and recall bias). Second, this study focused on specific demographic population (i.e., non-clinical, school-based students), and the generalizability of the results to other population should be cautious. Studies in other demographic population (i.e., psychiatric clinical population) are necessary to further confirm such longitudinal associations found in this study. Third, there may exist misclassification for depression as a source of measurement error considering that depression was measured by a self-administered epidemiological screening scale rather than clinical diagnosis to assess depression. Fourth, this study was restricted to two time points with 9-month interval. As we defined change in OSNA/depression (i.e., persistent ONSA/depression and remission from OSNA/depression) by comparing results of baseline and follow-up surveys that were conducted 9 months apart, we do not know whether OSNA/depression status changed or fluctuated during the 9-month period. Longitudinal studies with multiple observations and short time interval are necessary to capture the dynamic picture of these negative conditions. Fifth, considering that there is no available golden standard instrument and diagnostic criteria for OSNA, we used 10th decile of the OSNA scores at baseline to define OSNA cases following similar published study (Verkuijl et al., 2014). The sensitivity and specificity of such criterion for OSNA status is unclear and need to be evaluated in future research. However, the OSNA scale showed acceptable psychometric properties in this study and our previous studies. Sixth, the longitudinal associations between OSNA and depression were estimated separately using two subsamples. We believe that using pathological status as outcome rather than continuous scores could provide more meaningful explanation in epidemiological study. Cross-lagged structural equation modeling could be an alternative approach to explore causal directions in future longitudinal studies with three or more observations. In addition, our findings provide strong evidences of temporal associations (one important criterion for causal inference) between OSNA and depression. However, we could not rule out the possibility that a third variable not included in this study linked the longitudinal associations between OSNA and depression.

Conclusions

This study revealed a bidirectional association between OSNA and depression among adolescents, meaning that depression significantly contributes to the development of OSNA, and in turn, depressed individuals experience more deleterious effects from addictive online social networking use. More longitudinal studies with multiple observational time points and short-time interval are warranted for further confirmation of the findings from this study.

Authors’ contribution

J-BL, JTFL, PKHM, and X-FS conceived and designed the study. J-BL, J-CM, and Y-XC acquired the data. J-BL, JTFL, and PKHM performed the statistical analyses. J-BL, JTFL, PKHM, XZ, and AMSW drafted and revised the manuscript. All authors contributed to the interpretation of the results and critical revision of the manuscript for important intellectual content and approved the final version of the manuscript.

Conflict of interest

The authors declare no conflict of interest.

Acknowledgements

The authors would like to appreciate all participants and their families and schools for supporting this study.

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Dr. Zsolt Demetrovics
Institute of Psychology, ELTE Eötvös Loránd University
Address: Izabella u. 46. H-1064 Budapest, Hungary
Phone: +36-1-461-2681
E-mail: jba@ppk.elte.hu

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2023  
Web of Science  
Journal Impact Factor 6.6
Rank by Impact Factor Q1 (Psychiatry)
Journal Citation Indicator 1.59
Scopus  
CiteScore 12.3
CiteScore rank Q1 (Clinical Psychology)
SNIP 1.604
Scimago  
SJR index 2.188
SJR Q rank Q1

Journal of Behavioral Addictions
Publication Model Gold Open Access
Submission Fee none
Article Processing Charge 990 EUR/article for articles submitted after 30 April 2023 (850 EUR for articles submitted prior to this date)
Regional discounts on country of the funding agency World Bank Lower-middle-income economies: 50%
World Bank Low-income economies: 100%
Further Discounts Corresponding authors, affiliated to an EISZ member institution subscribing to the journal package of Akadémiai Kiadó: 100%.
Subscription Information Gold Open Access

Journal of Behavioral Addictions
Language English
Size A4
Year of
Foundation
2011
Volumes
per Year
1
Issues
per Year
4
Founder Eötvös Loránd Tudományegyetem
Founder's
Address
H-1053 Budapest, Hungary Egyetem tér 1-3.
Publisher Akadémiai Kiadó
Publisher's
Address
H-1117 Budapest, Hungary 1516 Budapest, PO Box 245.
Responsible
Publisher
Chief Executive Officer, Akadémiai Kiadó
ISSN 2062-5871 (Print)
ISSN 2063-5303 (Online)

Senior editors

Editor(s)-in-Chief: Zsolt DEMETROVICS

Assistant Editor(s): Csilla ÁGOSTON

Associate Editors

  • Stephanie ANTONS (Universitat Duisburg-Essen, Germany)
  • Joel BILLIEUX (University of Lausanne, Switzerland)
  • Beáta BŐTHE (University of Montreal, Canada)
  • Matthias BRAND (University of Duisburg-Essen, Germany)
  • Ruth J. van HOLST (Amsterdam UMC, The Netherlands)
  • Daniel KING (Flinders University, Australia)
  • Gyöngyi KÖKÖNYEI (ELTE Eötvös Loránd University, Hungary)
  • Ludwig KRAUS (IFT Institute for Therapy Research, Germany)
  • Marc N. POTENZA (Yale University, USA)
  • Hans-Jurgen RUMPF (University of Lübeck, Germany)

Editorial Board

  • Sophia ACHAB (Faculty of Medicine, University of Geneva, Switzerland)
  • Alex BALDACCHINO (St Andrews University, United Kingdom)
  • Judit BALÁZS (ELTE Eötvös Loránd University, Hungary)
  • Maria BELLRINGER (Auckland University of Technology, Auckland, New Zealand)
  • Henrietta BOWDEN-JONES (Imperial College, United Kingdom)
  • Damien BREVERS (University of Luxembourg, Luxembourg)
  • Wim VAN DEN BRINK (University of Amsterdam, The Netherlands)
  • Julius BURKAUSKAS (Lithuanian University of Health Sciences, Lithuania)
  • Gerhard BÜHRINGER (Technische Universität Dresden, Germany)
  • Silvia CASALE (University of Florence, Florence, Italy)
  • Luke CLARK (University of British Columbia, Vancouver, B.C., Canada)
  • Jeffrey L. DEREVENSKY (McGill University, Canada)
  • Geert DOM (University of Antwerp, Belgium)
  • Nicki DOWLING (Deakin University, Geelong, Australia)
  • Hamed EKHTIARI (University of Minnesota, United States)
  • Jon ELHAI (University of Toledo, Toledo, Ohio, USA)
  • Ana ESTEVEZ (University of Deusto, Spain)
  • Fernando FERNANDEZ-ARANDA (Bellvitge University Hospital, Barcelona, Spain)
  • Naomi FINEBERG (University of Hertfordshire, United Kingdom)
  • Sally GAINSBURY (The University of Sydney, Camperdown, NSW, Australia)
  • Belle GAVRIEL-FRIED (The Bob Shapell School of Social Work, Tel Aviv University, Israel)
  • Biljana GJONESKA (Macedonian Academy of Sciences and Arts, Republic of North Macedonia)
  • Marie GRALL-BRONNEC (University Hospital of Nantes, France)
  • Jon E. GRANT (University of Minnesota, USA)
  • Mark GRIFFITHS (Nottingham Trent University, United Kingdom)
  • Joshua GRUBBS (University of New Mexico, Albuquerque, NM, USA)
  • Anneke GOUDRIAAN (University of Amsterdam, The Netherlands)
  • Susumu HIGUCHI (National Hospital Organization Kurihama Medical and Addiction Center, Japan)
  • David HODGINS (University of Calgary, Canada)
  • Eric HOLLANDER (Albert Einstein College of Medicine, USA)
  • Zsolt HORVÁTH (Eötvös Loránd University, Hungary)
  • Susana JIMÉNEZ-MURCIA (Clinical Psychology Unit, Bellvitge University Hospital, Barcelona, Spain)
  • Yasser KHAZAAL (Geneva University Hospital, Switzerland)
  • Orsolya KIRÁLY (Eötvös Loránd University, Hungary)
  • Chih-Hung KO (Faculty of Medicine, College of Medicine, Kaohsiung Medical University, Taiwan)
  • Shane KRAUS (University of Nevada, Las Vegas, NV, USA)
  • Hae Kook LEE (The Catholic University of Korea, Republic of Korea)
  • Bernadette KUN (Eötvös Loránd University, Hungary)
  • Katerina LUKAVSKA (Charles University, Prague, Czech Republic)
  • Giovanni MARTINOTTI (‘Gabriele d’Annunzio’ University of Chieti-Pescara, Italy)
  • Gemma MESTRE-BACH (Universidad Internacional de la Rioja, La Rioja, Spain)
  • Astrid MÜLLER (Hannover Medical School, Germany)
  • Daniel Thor OLASON (University of Iceland, Iceland)
  • Ståle PALLESEN (University of Bergen, Norway)
  • Afarin RAHIMI-MOVAGHAR (Teheran University of Medical Sciences, Iran)
  • József RÁCZ (Hungarian Academy of Sciences, Hungary)
  • Michael SCHAUB (University of Zurich, Switzerland)
  • Marcantanio M. SPADA (London South Bank University, United Kingdom)
  • Daniel SPRITZER (Study Group on Technological Addictions, Brazil)
  • Dan J. STEIN (University of Cape Town, South Africa)
  • Sherry H. STEWART (Dalhousie University, Canada)
  • Attila SZABÓ (Eötvös Loránd University, Hungary)
  • Hermano TAVARES (Instituto de Psiquiatria do Hospital das Clínicas da Faculdade de Medicina, Universidade de São Paulo, São Paulo, Brazil)
  • Alexander E. VOISKOUNSKY (Moscow State University, Russia)
  • Aviv M. WEINSTEIN (Ariel University, Israel)
  • Anise WU (University of Macau, Macao, China)

 

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